Showing results (21-30 of 77) with videos related to
Sort By:
Pageof 8
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 22, 2023
Involvement of GABAergic Interneuron Subtypes in 4-Aminopyridine-Induced Seizure-Like Events in Mouse Entorhinal Cortex in VitroPaolo Scalmani, Rosina Paterra, Massimo Mantegazza, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 18, 2008
Self-limited hyperexcitability: functional effect of a familial hemiplegic migraine mutation of the Nav1.1 (SCN1A) Na+ channelSandrine Cestèle, Paolo Scalmani, Raffaella Rusconi, et al.Frontiers in Molecular Neuroscience|July 25, 2018
Gain of Function for the SCN1A/hNav1.1-L1670W Mutation Responsible for Familial Hemiplegic MigraineSandra Dhifallah, Eric Lancaster, Shana Merrill, et al.American Journal of Medical Genetics. Part A|September 19, 2009
Progressive neurocognitive decline in two children with Dravet syndrome, de novo SCN1A truncations and different epileptic phenotypesDaria Riva, Chiara Vago, Chiara Pantaleoni, et al.Neuropharmacology|January 17, 2020
Cholinergic modulation inhibits cortical spreading depression in mouse neocortex through activation of muscarinic receptors and decreased excitatory/inhibitory driveSarah Zerimech, Oana Chever, Paolo Scalmani, et al.Plos Computational Biology|July 27, 2021
Modeling NaV1.1/SCN1A sodium channel mutations in a microcircuit with realistic ion concentration dynamics suggests differential GABAergic mechanisms leading to hyperexcitability in epilepsy and hemiplegic migraineLouisiane Lemaire, Mathieu Desroches, Martin Krupa, et al.Neuroscience Letters|May 20, 2017
The impact of genetic and experimental studies on classification and therapy of the epilepsiesGiuliano Avanzini, Massimo Mantegazza, Benedetta Terragni, et al.Epilepsia|December 14, 2011
Pure haploinsufficiency for Dravet syndrome Na(V)1.1 (SCN1A) sodium channel truncating mutationsGiulia Bechi, Paolo Scalmani, Emanuele Schiavon, et al.Neurobiology of Disease|January 11, 2015
Rescuable folding defective NaV1.1 (SCN1A) mutants in epilepsy: properties, occurrence, and novel rescuing strategy with peptides targeted to the endoplasmic reticulumGiulia Bechi, Raffaella Rusconi, Sandrine Cestèle, et al.Physiological Reviews|August 11, 2022
Developmental and epileptic encephalopathies: from genetic heterogeneity to phenotypic continuumRenzo Guerrini, Valerio Conti, Massimo Mantegazza, et al.Pageof 8