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The Journal of Clinical Endocrinology and Metabolism|January 15, 2002
The RET/PTC oncogene is frequently activated in oncocytic thyroid tumors (Hurthle cell adenomas and carcinomas), but not in oncocytic hyperplastic lesionsGennaro Chiappetta, Paolo Toti, Francesco Cetta, et al.
Sleep & Breathing = Schlaf & Atmung|December 7, 2013
Prevalence and clinical correlates of sleep disordered breathing in myotonic dystrophy types 1 and 2Maria Laura Ester Bianchi, Anna Losurdo, Chiara Di Blasi, et al.
Oncogene|March 16, 2002
Glial cell line-derived neurotrophic factor induces proliferative inhibition of NT2/D1 cells through RET-mediated up-regulation of the cyclin-dependent kinase inhibitor p27(kip1)Gustavo Baldassarre, Paola Bruni, Angelo Boccia, et al.
Endocrine-Related Cancer|February 13, 2019
Association between DNA methylation profile and malignancy in follicular-patterned thyroid neoplasmsOrnella Affinito, Paolo Salerno, Alfonso D'Alessio, et al.
The Journal of Clinical Endocrinology and Metabolism|October 9, 2004
Analysis of BRAF point mutation and RET/PTC rearrangement refines the fine-needle aspiration diagnosis of papillary thyroid carcinomaGiuliana Salvatore, Riccardo Giannini, Pinuccia Faviana, et al.
The Journal of Clinical Endocrinology and Metabolism|March 11, 2011
TWIST1 plays a pleiotropic role in determining the anaplastic thyroid cancer phenotypePaolo Salerno, Ginesa Garcia-Rostan, Sara Piccinin, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 4, 2012
Activation of the mTOR pathway in primary medullary thyroid carcinoma and lymph node metastasesAnna Tamburrino, Alfredo A Molinolo, Paolo Salerno, et al.
Angewandte Chemie (International Ed. in English)|July 2, 2015
Fragment-Based Discovery of a Dual pan-RET/VEGFR2 Kinase Inhibitor Optimized for Single-Agent PolypharmacologyBrendan Frett, Francesca Carlomagno, Maria Luisa Moccia, et al.
Journal of Neurology|December 25, 2012
Molecular, clinical, and muscle studies in myotonic dystrophy type 1 (DM1) associated with novel variant CCG expansionsMassimo Santoro, Marcella Masciullo, Roberta Pietrobono, et al.
The American Journal of Pathology|July 28, 2004
The oncogenic activity of RET point mutants for follicular thyroid cells may account for the occurrence of papillary thyroid carcinoma in patients affected by familial medullary thyroid carcinomaRosa Marina Melillo, Anna Maria Cirafici, Valentina De Falco, et al.
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