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The International Journal of Biochemistry & Cell Biology|December 18, 2018
The paroxysmal depolarization shift in epilepsy researchMatej Hotka, Helmut KubistaBio-Protocol|January 26, 2019
Detection of Ligand-binding to Membrane Proteins by Capacitance MeasurementsVerena Burtscher, Matej Hotka, Walter SandtnerInternational Journal of Molecular Sciences|February 1, 2019
The Paroxysmal Depolarization Shift: Reconsidering Its Role in Epilepsy, Epileptogenesis and BeyondHelmut Kubista, Stefan Boehm, Matej HotkaBiophysical Journal|August 15, 2019
An Electrophysiological Approach to Measure Changes in the Membrane Surface Potential in Real TimeVerena Burtscher, Matej Hotka, Michael Freissmuth, et al.Elife|April 27, 2018
A label-free approach to detect ligand binding to cell surface proteins in real timeVerena Burtscher, Matej Hotka, Yang Li, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 23, 2022
Glycerol-3-Phosphate Shuttle Is a Backup System Securing Metabolic Flexibility in NeuronsAnkit Dhoundiyal, Vanessa Goeschl, Stefan Boehm, et al.Plos One|June 7, 2013
Quantitative analysis of calcium spikes in noisy fluorescent backgroundRadoslav Janicek, Matej Hotka, Alexandra Zahradníková, et al.Neuropharmacology|August 8, 2025
IP<sub>3</sub>-mediated Ca<sup>2+</sup> transfer from ER to mitochondria stimulates ATP synthesis in primary hippocampal neuronsAnkit Dhoundiyal, Vanessa Goeschl, Stefan Boehm, et al.Epilepsia|March 16, 2017
Ca<sub>v</sub> 1.3 channels play a crucial role in the formation of paroxysmal depolarization shifts in cultured hippocampal neuronsVictoria Stiglbauer, Matej Hotka, Manuel Ruiß, et al.Science Signaling|February 13, 2020
L-type Ca<sup>2+</sup> channel-mediated Ca<sup>2+</sup> influx adjusts neuronal mitochondrial function to physiological and pathophysiological conditionsMatej Hotka, Michal Cagalinec, Karlheinz Hilber, et al.Pageof 2