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Behavioural Brain Research|September 22, 2009
Behavioral phenotyping of heterozygous acetylcholinesterase knockout (AChE+/-) mice showed no memory enhancement but hyposensitivity to amnesic drugsJulie Espallergues, Laurie Galvan, Florence Sabatier, et al.
Journal of Huntington'S Disease|July 27, 2014
Functional Differences Between Direct and Indirect Striatal Output Pathways in Huntington's DiseaseLaurie Galvan, Véronique M André, Elizabeth A Wang, et al.
Scientific Reports|July 11, 2023
Mice prenatally exposed to valproic acid do not show autism-related disorders when fed with polyunsaturated fatty acid-enriched dietsValentine Turpin, Maud Schaffhauser, Mathieu Thabault, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 26, 2013
Multiple sources of striatal inhibition are differentially affected in Huntington's disease mouse modelsCarlos Cepeda, Laurie Galvan, Sandra M Holley, et al.
Neurobiology of Aging|January 27, 2015
Striatal long noncoding RNA Abhd11os is neuroprotective against an N-terminal fragment of mutant huntingtin in vivoLaetitia Francelle, Laurie Galvan, Marie-Claude Gaillard, et al.
Human Molecular Genetics|November 16, 2014
Loss of the thyroid hormone-binding protein Crym renders striatal neurons more vulnerable to mutant huntingtin in Huntington's diseaseLaetitia Francelle, Laurie Galvan, Marie-Claude Gaillard, et al.
Brain : a Journal of Neurology|February 26, 2016
CYP46A1, the rate-limiting enzyme for cholesterol degradation, is neuroprotective in Huntington's diseaseLydie Boussicault, Sandro Alves, Antonin Lamazière, et al.
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