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Clinical Journal of the American Society of Nephrology : CJASN|January 21, 2026
A New Hope for Treating Podocytopathies - Emerging Role of Anti-Nephrin AntibodyNastassia Liaukouskaya, Felicitas E Hengel, Anne Mühlig, et al.Autophagy|April 26, 2013
The class III phosphatidylinositol 3-kinase PIK3C3/VPS34 regulates endocytosis and autophagosome-autolysosome formation in podocytesWibke Bechtel, Martin Helmstädter, Jan Balica, et al.Nature Reviews. Nephrology|August 2, 2016
Roles of mTOR complexes in the kidney: implications for renal disease and transplantationDaniel Fantus, Natasha M Rogers, Florian Grahammer, et al.Plos One|August 23, 2011
Functional and spatial analysis of C. elegans SYG-1 and SYG-2, orthologs of the Neph/nephrin cell adhesion module directing selective synaptogenesisNicola Wanner, Foteini Noutsou, Ralf Baumeister, et al.American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation|June 24, 2014
Hantavirus infection with severe proteinuria and podocyte foot-process effacementChristopher Boehlke, Björn Hartleben, Tobias B Huber, et al.International Journal of Molecular Sciences|March 13, 2024
Cisplatin Nephrotoxicity Is Critically Mediated by the Availability of BECLIN1Tillmann Bork, Camila Hernando-Erhard, Wei Liang, et al.Pediatric Nephrology (Berlin, Germany)|April 23, 2026
Anti-nephrin antibodies in pediatric nephrotic syndrome: a new paradigmAnne K Mühlig, Felicitas E Hengel, Jun Oh, et al.Journal of Immunotherapy (Hagerstown, Md. : 1997)|October 1, 2019
Severe Acute Kidney Injury Due to Nivolumab/Ipilimumab-induced Granulomatosis and Fibrinoid Vascular NecrosisFermin Person, Tuhama Chahoud-Schriefer, Wilfrid Fehrle, et al.Frontiers in Physiology|October 13, 2022
A high-throughput drug discovery pipeline to optimize kidney normothermic machine perfusionSmilla Hofmann, Florian Grahammer, Ilka Edenhofer, et al.American Journal of Physiology. Renal Physiology|January 14, 2011
GSK3β inactivation in podocytes results in decreased phosphorylation of p70S6K accompanied by cytoskeletal rearrangements and inhibited motilityBritta George, Beate Vollenbröker, Moin A Saleem, et al.Pageof 37