Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Matija Hedl

Showing results (1-10 of 39) with videos related to

Pageof 4
Sort By:
Gut|July 29, 2015
A <i>TPL2 (MAP3K8)</i> disease-risk polymorphism increases TPL2 expression thereby leading to increased pattern recognition receptor-initiated caspase-1 and caspase-8 activation, signalling and cytokine secretionMatija Hedl, Clara Abraham
Gastroenterology|September 13, 2012
Nod2-induced autocrine interleukin-1 alters signaling by ERK and p38 to differentially regulate secretion of inflammatory cytokinesMatija Hedl, Clara Abraham
The Journal of Biological Chemistry|June 11, 2011
Distinct roles for Nod2 protein and autocrine interleukin-1beta in muramyl dipeptide-induced mitogen-activated protein kinase activation and cytokine secretion in human macrophagesMatija Hedl, Clara Abraham
Gastroenterology|September 22, 2010
Secretory mediators regulate Nod2-induced tolerance in human macrophagesMatija Hedl, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|May 1, 2012
IRF5 risk polymorphisms contribute to interindividual variance in pattern recognition receptor-mediated cytokine secretion in human monocyte-derived cellsMatija Hedl, Clara Abraham
Proceedings of the National Academy of Sciences of the United States of America|September 9, 2014
A TNFSF15 disease-risk polymorphism increases pattern-recognition receptor-induced signaling through caspase-8-induced IL-1Matija Hedl, Clara Abraham
American Journal of Physiology. Gastrointestinal and Liver Physiology|January 5, 2013
NLRP1 and NLRP3 inflammasomes are essential for distinct outcomes of decreased cytokines but enhanced bacterial killing upon chronic Nod2 stimulationMatija Hedl, Clara Abraham
Protein Science : a Publication of the Protein Society|May 21, 2004
Inhibition of the class II HMG-CoA reductase of Pseudomonas mevaloniiMatija Hedl, Victor W Rodwell
Protein Science : a Publication of the Protein Society|February 10, 2004
Enterococcus faecalis mevalonate kinaseMatija Hedl, Victor W Rodwell
Journal of Immunology (Baltimore, Md. : 1950)|May 21, 2014
The IL18RAP region disease polymorphism decreases IL-18RAP/IL-18R1/IL-1R1 expression and signaling through innate receptor-initiated pathwaysMatija Hedl, Shasha Zheng, Clara Abraham
Pageof 4

Showing results (1-10 of 39) with videos related to

Sort By:
Pageof 4
Gut|July 29, 2015
A <i>TPL2 (MAP3K8)</i> disease-risk polymorphism increases TPL2 expression thereby leading to increased pattern recognition receptor-initiated caspase-1 and caspase-8 activation, signalling and cytokine secretionMatija Hedl, Clara Abraham
Gastroenterology|September 13, 2012
Nod2-induced autocrine interleukin-1 alters signaling by ERK and p38 to differentially regulate secretion of inflammatory cytokinesMatija Hedl, Clara Abraham
The Journal of Biological Chemistry|June 11, 2011
Distinct roles for Nod2 protein and autocrine interleukin-1beta in muramyl dipeptide-induced mitogen-activated protein kinase activation and cytokine secretion in human macrophagesMatija Hedl, Clara Abraham
Gastroenterology|September 22, 2010
Secretory mediators regulate Nod2-induced tolerance in human macrophagesMatija Hedl, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|May 1, 2012
IRF5 risk polymorphisms contribute to interindividual variance in pattern recognition receptor-mediated cytokine secretion in human monocyte-derived cellsMatija Hedl, Clara Abraham
Proceedings of the National Academy of Sciences of the United States of America|September 9, 2014
A TNFSF15 disease-risk polymorphism increases pattern-recognition receptor-induced signaling through caspase-8-induced IL-1Matija Hedl, Clara Abraham
American Journal of Physiology. Gastrointestinal and Liver Physiology|January 5, 2013
NLRP1 and NLRP3 inflammasomes are essential for distinct outcomes of decreased cytokines but enhanced bacterial killing upon chronic Nod2 stimulationMatija Hedl, Clara Abraham
Protein Science : a Publication of the Protein Society|May 21, 2004
Inhibition of the class II HMG-CoA reductase of Pseudomonas mevaloniiMatija Hedl, Victor W Rodwell
Protein Science : a Publication of the Protein Society|February 10, 2004
Enterococcus faecalis mevalonate kinaseMatija Hedl, Victor W Rodwell
Journal of Immunology (Baltimore, Md. : 1950)|May 21, 2014
The IL18RAP region disease polymorphism decreases IL-18RAP/IL-18R1/IL-1R1 expression and signaling through innate receptor-initiated pathwaysMatija Hedl, Shasha Zheng, Clara Abraham
Pageof 4