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Matilde Said

Showing results (1-10 of 21) with videos related to

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Naunyn-Schmiedeberg'S Archives of Pharmacology|April 1, 2006
beta 2-Adrenergic stimulation is involved in the contractile dysfunction of the stunned heartLeticia Vittone, Matilde Said, Alicia Mattiazzi
Cardiovascular Research|October 21, 2025
Kinases go where the function callsMatilde Said, Alicia Mattiazzi, Cecilia Mundiña-Weilenmann
Pflugers Archiv : European Journal of Physiology|October 2, 2002
The relative relevance of phosphorylation of the Thr(17) residue of phospholamban is different at different levels of beta-adrenergic stimulationMatilde Said, Cecilia Mundiña-Weilenmann, Leticia Vittone, et al.
Molecular and Cellular Biochemistry|October 28, 2003
Phospholamban phosphorylation in ischemia-reperfused heart. Effect of pacing during ischemia and response to a beta-adrenergic challengeCecilia Mundiña-Weilenmann, Matilde Said, Leticia Vittone, et al.
Journal of Molecular and Cellular Cardiology|January 29, 2002
Time course and mechanisms of phosphorylation of phospholamban residues in ischemia-reperfused rat hearts. Dissociation of phospholamban phosphorylation pathwaysLeticia Vittone, Cecilia Mundiña-Weilenmann, Matilde Said, et al.
European Journal of Pharmacology|December 3, 2021
Pharmacological inhibition of translocon is sufficient to alleviate endoplasmic reticulum stress and improve Ca<sup>2+</sup> handling and contractile recovery of stunned myocardiumJuan Ignacio Elio Mariángelo, Carlos Alfredo Valverde, Leticia Vittone, et al.
Cardiovascular Research|March 17, 2005
Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosisCecilia Mundiña-Weilenmann, Paola Ferrero, Matilde Said, et al.
Journal of Molecular and Cellular Cardiology|April 30, 2013
Role of CaMKII in post acidosis arrhythmias: a simulation study using a human myocyte modelElena C Lascano, Matilde Said, Leticia Vittone, et al.
Journal of Molecular and Cellular Cardiology|January 15, 2014
Stimulation of NOX2 in isolated hearts reversibly sensitizes RyR2 channels to activation by cytoplasmic calciumPaulina Donoso, José Pablo Finkelstein, Luis Montecinos, et al.
Acta Physiologica (Oxford, England)|March 27, 2023
Prolonged Ca<sup>2+</sup> release refractoriness and T-tubule disruption as determinants of increased propensity to cardiac alternans in the hypertensive heart diseaseJuan Ignacio Elio Mariángelo, Gabriel Darío Di Marzio, Luis Alberto Gonano, et al.
Pageof 3

Showing results (1-10 of 21) with videos related to

Sort By:
Pageof 3
Naunyn-Schmiedeberg'S Archives of Pharmacology|April 1, 2006
beta 2-Adrenergic stimulation is involved in the contractile dysfunction of the stunned heartLeticia Vittone, Matilde Said, Alicia Mattiazzi
Cardiovascular Research|October 21, 2025
Kinases go where the function callsMatilde Said, Alicia Mattiazzi, Cecilia Mundiña-Weilenmann
Pflugers Archiv : European Journal of Physiology|October 2, 2002
The relative relevance of phosphorylation of the Thr(17) residue of phospholamban is different at different levels of beta-adrenergic stimulationMatilde Said, Cecilia Mundiña-Weilenmann, Leticia Vittone, et al.
Molecular and Cellular Biochemistry|October 28, 2003
Phospholamban phosphorylation in ischemia-reperfused heart. Effect of pacing during ischemia and response to a beta-adrenergic challengeCecilia Mundiña-Weilenmann, Matilde Said, Leticia Vittone, et al.
Journal of Molecular and Cellular Cardiology|January 29, 2002
Time course and mechanisms of phosphorylation of phospholamban residues in ischemia-reperfused rat hearts. Dissociation of phospholamban phosphorylation pathwaysLeticia Vittone, Cecilia Mundiña-Weilenmann, Matilde Said, et al.
European Journal of Pharmacology|December 3, 2021
Pharmacological inhibition of translocon is sufficient to alleviate endoplasmic reticulum stress and improve Ca<sup>2+</sup> handling and contractile recovery of stunned myocardiumJuan Ignacio Elio Mariángelo, Carlos Alfredo Valverde, Leticia Vittone, et al.
Cardiovascular Research|March 17, 2005
Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosisCecilia Mundiña-Weilenmann, Paola Ferrero, Matilde Said, et al.
Journal of Molecular and Cellular Cardiology|April 30, 2013
Role of CaMKII in post acidosis arrhythmias: a simulation study using a human myocyte modelElena C Lascano, Matilde Said, Leticia Vittone, et al.
Journal of Molecular and Cellular Cardiology|January 15, 2014
Stimulation of NOX2 in isolated hearts reversibly sensitizes RyR2 channels to activation by cytoplasmic calciumPaulina Donoso, José Pablo Finkelstein, Luis Montecinos, et al.
Acta Physiologica (Oxford, England)|March 27, 2023
Prolonged Ca<sup>2+</sup> release refractoriness and T-tubule disruption as determinants of increased propensity to cardiac alternans in the hypertensive heart diseaseJuan Ignacio Elio Mariángelo, Gabriel Darío Di Marzio, Luis Alberto Gonano, et al.
Pageof 3