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Archives of Biochemistry and Biophysics|December 20, 2011
Mitochondrial calpain 10 is degraded by Lon protease after oxidant injuryMatthew A Smith, Rick G SchnellmannCardiovascular Research|May 15, 2012
Calpains, mitochondria, and apoptosisMatthew A Smith, Rick G SchnellmannArchives of Biochemistry and Biophysics|May 10, 2012
Loss of calpain 10 causes mitochondrial dysfunction during chronic hyperglycemiaMatthew A Smith, Marisa D Covington, Rick G SchnellmannThe Journal of Pharmacology and Experimental Therapeutics|June 28, 2006
A death-promoting role for extracellular signal-regulated kinaseShougang Zhuang, Rick G SchnellmannThe Journal of Pharmacology and Experimental Therapeutics|December 20, 2002
Calpain mediates progressive plasma membrane permeability and proteolysis of cytoskeleton-associated paxillin, talin, and vinculin during renal cell deathXiuli Liu, Rick G SchnellmannNature Reviews. Nephrology|August 15, 2017
Mitochondrial energetics in the kidneyPallavi Bhargava, Rick G SchnellmannThe Journal of Pharmacology and Experimental Therapeutics|April 19, 2005
Suramin promotes proliferation and scattering of renal epithelial cellsShougang Zhuang, Rick G SchnellmannAmerican Journal of Physiology. Renal Physiology|December 14, 2011
Persistent disruption of mitochondrial homeostasis after acute kidney injuryJason A Funk, Rick G SchnellmannThe Journal of Pharmacology and Experimental Therapeutics|October 28, 2017
Extracellular Signal-Regulated Kinase 1/2 Regulates Mouse Kidney Injury Molecule-1 Expression Physiologically and Following Ischemic and Septic Renal InjuryJustin B Collier, Rick G SchnellmannBiochemical and Biophysical Research Communications|December 7, 2010
Visualization and quantification of endoplasmic reticulum Ca2+ in renal cells using confocal microscopy and Fluo5FAndre C Eaddy, Rick G SchnellmannPageof 32