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Biophysical Journal|October 22, 2020
Edge Strand Dissociation and Conformational Changes in Transthyretin under Amyloidogenic ConditionsMatthew C Childers, Valerie DaggettProtein Science : a Publication of the Protein Society|November 15, 2020
Conserved patterns and interactions in the unfolding transition state across SH3 domain structural homologuesCullen Demakis, Matthew C Childers, Valerie DaggettBiophysical Journal|February 27, 2026
In silico conformational dynamics of the α-actinin-2 actin-binding domain upon phosphorylationHelene Tigro, Matthew C Childers, Michael Regnier, et al.The Journal of General Physiology|April 12, 2024
Myosin's powerstroke transitions define atomic scale movement of cardiac thin filament tropomyosinMichael J Rynkiewicz, Matthew C Childers, Olga E Karpicheva, et al.Biorxiv : the Preprint Server for Biology|June 12, 2026
The hypertrophic cardiomyopathy mutation G768R makes cardiac myosin a high duty ratio motorDivya Pathak, Tugba N Ozturk, Aminah Dawood, et al.Biorxiv : the Preprint Server for Biology|March 11, 2024
Hypertrophic cardiomyopathy mutations Y115H and E497D disrupt the folded-back state of human beta-cardiac myosin allostericallyNeha Nandwani, Debanjan Bhowmik, Matthew C Childers, et al.Proceedings of the National Academy of Sciences of the United States of America|August 22, 2024
Multiscale modeling shows how 2'-deoxy-ATP rescues ventricular function in heart failureAbigail E Teitgen, Marcus T Hock, Kimberly J McCabe, et al.Elife|May 3, 2022
Post-translational modification patterns on β-myosin heavy chain are altered in ischemic and nonischemic human heartsMaicon Landim-Vieira, Matthew C Childers, Amanda L Wacker, et al.Journal of Molecular and Cellular Cardiology|October 30, 2025
Molecular mechanisms of altered contraction with the β-myosin R403Q mutation in porcine ventricular muscle and a human stem cell-derived cardiomyocyte modelSonette Steczina, Saffie Mohran, Matthew C Childers, et al.Biorxiv : the Preprint Server for Biology|September 15, 2025
Molecular mechanisms of altered contraction with the β-myosin R403Q mutation in porcine ventricular muscle and a human stem cell-derived cardiomyocyte modelSonette Steczina, Saffie Mohran, Matthew C Childers, et al.Pageof 2