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Matthew K Lancaster

Showing results (1-10 of 16) with videos related to

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Mechanisms of Ageing and Development|May 10, 2015
Progressive age-associated activation of JNK associates with conduction disruption in the aged atriumSandra A Jones, Matthew K Lancaster
Experimental Gerontology|April 10, 2021
Reduced cardiac response to the adrenergic system is a key limiting factor for physical capacity in old ageLuke A Howlett, Matthew K Lancaster
Expert Review of Cardiovascular Therapy|September 1, 2011
Aging is a primary risk factor for cardiac arrhythmias: disruption of intracellular Ca2+ regulation as a key suspectFiona Hatch, Matthew K Lancaster, Sandra A Jones
Current Vascular Pharmacology|January 7, 2022
Pharmacy and Exercise as Complimentary Partners for Successful Cardiovascular AgeingLuke A Howlett, Sandra A Jones, Matthew K Lancaster
Circulation|March 7, 2007
Declining into failure: the age-dependent loss of the L-type calcium channel within the sinoatrial nodeSandra A Jones, Mark R Boyett, Matthew K Lancaster
The Journal of Physiology|February 25, 2026
An age-associated decline in the role of the sarcoplasmic reticulum and associated calcium-handling proteins sets the pace for sinoatrial node functionSandra A Jones, Fiona S Godbeer, Matthew K Lancaster
The Journal of Physiology|August 17, 2004
Ageing-related changes of connexins and conduction within the sinoatrial nodeSandra A Jones, Matthew K Lancaster, Mark R Boyett
Journal of Cardiovascular Electrophysiology|December 6, 2018
K<sub>2p</sub> 3.1 protein is expressed as a transmural gradient across the rat left ventricular free wallSandra A Jones, Richard D Walton, Michael Morton, et al.
The Journal of Physiology|January 16, 2004
Intracellular Ca2+ and pacemaking within the rabbit sinoatrial node: heterogeneity of role and controlMatthew K Lancaster, Sandra A Jones, Simon M Harrison, et al.
Physiological Reports|July 26, 2023
Ionic current changes underlying action potential repolarization responses to physiological pacing and adrenergic stimulation in adult rat ventricular myocytesLuke A Howlett, Harley Stevenson-Cocks, Michael A Colman, et al.
Pageof 2

Showing results (1-10 of 16) with videos related to

Sort By:
Pageof 2
Mechanisms of Ageing and Development|May 10, 2015
Progressive age-associated activation of JNK associates with conduction disruption in the aged atriumSandra A Jones, Matthew K Lancaster
Experimental Gerontology|April 10, 2021
Reduced cardiac response to the adrenergic system is a key limiting factor for physical capacity in old ageLuke A Howlett, Matthew K Lancaster
Expert Review of Cardiovascular Therapy|September 1, 2011
Aging is a primary risk factor for cardiac arrhythmias: disruption of intracellular Ca2+ regulation as a key suspectFiona Hatch, Matthew K Lancaster, Sandra A Jones
Current Vascular Pharmacology|January 7, 2022
Pharmacy and Exercise as Complimentary Partners for Successful Cardiovascular AgeingLuke A Howlett, Sandra A Jones, Matthew K Lancaster
Circulation|March 7, 2007
Declining into failure: the age-dependent loss of the L-type calcium channel within the sinoatrial nodeSandra A Jones, Mark R Boyett, Matthew K Lancaster
The Journal of Physiology|February 25, 2026
An age-associated decline in the role of the sarcoplasmic reticulum and associated calcium-handling proteins sets the pace for sinoatrial node functionSandra A Jones, Fiona S Godbeer, Matthew K Lancaster
The Journal of Physiology|August 17, 2004
Ageing-related changes of connexins and conduction within the sinoatrial nodeSandra A Jones, Matthew K Lancaster, Mark R Boyett
Journal of Cardiovascular Electrophysiology|December 6, 2018
K<sub>2p</sub> 3.1 protein is expressed as a transmural gradient across the rat left ventricular free wallSandra A Jones, Richard D Walton, Michael Morton, et al.
The Journal of Physiology|January 16, 2004
Intracellular Ca2+ and pacemaking within the rabbit sinoatrial node: heterogeneity of role and controlMatthew K Lancaster, Sandra A Jones, Simon M Harrison, et al.
Physiological Reports|July 26, 2023
Ionic current changes underlying action potential repolarization responses to physiological pacing and adrenergic stimulation in adult rat ventricular myocytesLuke A Howlett, Harley Stevenson-Cocks, Michael A Colman, et al.
Pageof 2