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Matthew P Frosch

Showing results (61-70 of 325) with videos related to

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The New England Journal of Medicine|April 9, 2010
Case records of the Massachusetts General Hospital. Case 10-2010. A 37-year-old woman with weakness and a mass in the brainDaniel P Cahill, Fred G Barker, Kenneth R Davis, et al.
Journal of Neuroimaging : Official Journal of the American Society of Neuroimaging|August 20, 2016
Susceptibility Etching on MRI in Patients with MicroangiopathyJennifer Chang, Keerthi Arani, Sheena Chew, et al.
Anesthesiology|May 13, 2010
Posterior circulation stroke after C1-C2 intraarticular facet steroid injection: evidence for diffuse microvascular injuryBrian L Edlow, Brian J Wainger, Matthew P Frosch, et al.
The American Journal of Pathology|August 8, 2012
The synaptic accumulation of hyperphosphorylated tau oligomers in Alzheimer disease is associated with dysfunction of the ubiquitin-proteasome systemHwan-Ching Tai, Alberto Serrano-Pozo, Tadafumi Hashimoto, et al.
Neurobiology of Disease|March 26, 2021
Impairment of visual cortical plasticity by amyloid-beta speciesChristopher M William, Matthew A Stern, Xuewei Pei, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|January 29, 2020
In vivo characterization of spontaneous microhemorrhage formation in mice with cerebral amyloid angiopathySusanne J van Veluw, Matthew P Frosch, Ashley A Scherlek, et al.
Cerebellum (London, England)|August 24, 2012
Spinocerebellar ataxia type 7: clinical course, phenotype-genotype correlations, and neuropathologyLaura C Horton, Matthew P Frosch, Mark G Vangel, et al.
Neurobiology of Disease|April 7, 2007
Effects of gender on nigral gene expression and parkinson diseaseIppolita Cantuti-Castelvetri, Christine Keller-McGandy, Bérengère Bouzou, et al.
Acta Neuropathologica Communications|October 22, 2014
Frequent and symmetric deposition of misfolded tau oligomers within presynaptic and postsynaptic terminals in Alzheimer's diseaseHwan-Ching Tai, Bo Y Wang, Alberto Serrano-Pozo, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 18, 2005
Familial Alzheimer's disease presenilin 1 mutations cause alterations in the conformation of presenilin and interactions with amyloid precursor proteinOksana Berezovska, Alberto Lleo, Lauren D Herl, et al.
Pageof 33

Showing results (61-70 of 325) with videos related to

Sort By:
Pageof 33
The New England Journal of Medicine|April 9, 2010
Case records of the Massachusetts General Hospital. Case 10-2010. A 37-year-old woman with weakness and a mass in the brainDaniel P Cahill, Fred G Barker, Kenneth R Davis, et al.
Journal of Neuroimaging : Official Journal of the American Society of Neuroimaging|August 20, 2016
Susceptibility Etching on MRI in Patients with MicroangiopathyJennifer Chang, Keerthi Arani, Sheena Chew, et al.
Anesthesiology|May 13, 2010
Posterior circulation stroke after C1-C2 intraarticular facet steroid injection: evidence for diffuse microvascular injuryBrian L Edlow, Brian J Wainger, Matthew P Frosch, et al.
The American Journal of Pathology|August 8, 2012
The synaptic accumulation of hyperphosphorylated tau oligomers in Alzheimer disease is associated with dysfunction of the ubiquitin-proteasome systemHwan-Ching Tai, Alberto Serrano-Pozo, Tadafumi Hashimoto, et al.
Neurobiology of Disease|March 26, 2021
Impairment of visual cortical plasticity by amyloid-beta speciesChristopher M William, Matthew A Stern, Xuewei Pei, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|January 29, 2020
In vivo characterization of spontaneous microhemorrhage formation in mice with cerebral amyloid angiopathySusanne J van Veluw, Matthew P Frosch, Ashley A Scherlek, et al.
Cerebellum (London, England)|August 24, 2012
Spinocerebellar ataxia type 7: clinical course, phenotype-genotype correlations, and neuropathologyLaura C Horton, Matthew P Frosch, Mark G Vangel, et al.
Neurobiology of Disease|April 7, 2007
Effects of gender on nigral gene expression and parkinson diseaseIppolita Cantuti-Castelvetri, Christine Keller-McGandy, Bérengère Bouzou, et al.
Acta Neuropathologica Communications|October 22, 2014
Frequent and symmetric deposition of misfolded tau oligomers within presynaptic and postsynaptic terminals in Alzheimer's diseaseHwan-Ching Tai, Bo Y Wang, Alberto Serrano-Pozo, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 18, 2005
Familial Alzheimer's disease presenilin 1 mutations cause alterations in the conformation of presenilin and interactions with amyloid precursor proteinOksana Berezovska, Alberto Lleo, Lauren D Herl, et al.
Pageof 33