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Neuroscience Letters|February 15, 2020
Synaptotagmin: Mechanisms of an electrostatic switchMatthew R Bowers, Noreen E Reist
Plos One|February 8, 2020
The C2A domain of synaptotagmin is an essential component of the calcium sensor for synaptic transmissionMatthew R Bowers, Noreen E Reist
Plos One|May 15, 2020
The role of the C2A domain of synaptotagmin 1 in asynchronous neurotransmitter releaseMallory C Shields, Matthew R Bowers, Hannah L Kramer, et al.
Traffic (Copenhagen, Denmark)|September 18, 2008
The Drosophila epsin 1 is required for ubiquitin-dependent synaptic growth and function but not for synaptic vesicle recyclingHong Bao, Noreen E Reist, Bing Zhang
The Journal of Comparative Neurology|March 11, 2006
Drosophila synaptotagmin I null mutants show severe alterations in vesicle populations but calcium-binding motif mutants do notCarin A Loewen, Suzanne M Royer, Noreen E Reist
Journal of Neurophysiology|November 3, 2006
Nerve-evoked synchronous release and high K+ -induced quantal events are regulated separately by synaptotagmin I at Drosophila neuromuscular junctionsTakuya Tamura, Jiamei Hou, Noreen E Reist, et al.
Genesis (New York, N.Y. : 2000)|April 10, 2009
Synaptotagmin I stabilizes synaptic vesicles via its C(2)A polylysine motifKimberly E Mace, Laurie M Biela, Anastasia G Sares, et al.
Plos One|September 28, 2017
Drosophila studies support a role for a presynaptic synaptotagmin mutation in a human congenital myasthenic syndromeMallory C Shields, Matthew R Bowers, McKenzie M Fulcer, et al.
Molecular Biology of the Cell|September 22, 2006
C2B polylysine motif of synaptotagmin facilitates a Ca2+-independent stage of synaptic vesicle priming in vivoCarin A Loewen, Soo-Min Lee, Yeon-Kyun Shin, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 25, 2008
Ca2+-dependent, phospholipid-binding residues of synaptotagmin are critical for excitation-secretion coupling in vivoBrie E Paddock, Amelia R Striegel, Enfu Hui, et al.
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