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Science Translational Medicine|November 25, 2016
Two tissue-resident progenitor lineages drive distinct phenotypes of heterotopic ossificationDevaveena Dey, Jana Bagarova, Sarah J Hatsell, et al.The Journal of Clinical Investigation|May 5, 2022
Anti-ACVR1 antibodies exacerbate heterotopic ossification in fibrodysplasia ossificans progressiva (FOP) by activating FOP-mutant ACVR1Senem Aykul, Lily Huang, Lili Wang, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|August 8, 2017
The Expansion of Heterotopic Bone in Fibrodysplasia Ossificans Progressiva Is Activin A-DependentJaymin Upadhyay, LiQin Xie, Lily Huang, et al.Science Translational Medicine|September 4, 2015
ACVR1R206H receptor mutation causes fibrodysplasia ossificans progressiva by imparting responsiveness to activin ASarah J Hatsell, Vincent Idone, Dana M Alessi Wolken, et al.Proceedings of the National Academy of Sciences of the United States of America|August 7, 2013
Conditionals by inversion provide a universal method for the generation of conditional allelesAris N Economides, David Frendewey, Peter Yang, et al.Genome Research|October 4, 2003
Development of human protein reference database as an initial platform for approaching systems biology in humansSuraj Peri, J Daniel Navarro, Ramars Amanchy, et al.Pageof 7