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Matthew T Dickerson

Showing results (11-20 of 29) with videos related to

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Molecular Metabolism|February 7, 2018
TALK-1 reduces delta-cell endoplasmic reticulum and cytoplasmic calcium levels limiting somatostatin secretionNicholas C Vierra, Matthew T Dickerson, Kelli L Jordan, et al.
Molecular Metabolism|December 6, 2025
Glycolytic activation of β-cell Na<sup>+</sup>/K<sup>+</sup>-ATPases containing β1-subunits accelerates Na<sup>+</sup> extrusion, prolonging the duration of Ca<sup>2+</sup> oscillations but decreasing insulin secretionMatthew T Dickerson, Prasanna K Dadi, Reagan P McDevitt, et al.
American Journal of Physiology. Endocrinology and Metabolism|January 30, 2019
Glucose-mediated inhibition of calcium-activated potassium channels limits α-cell calcium influx and glucagon secretionMatthew T Dickerson, Prasanna K Dadi, Molly K Altman, et al.
Diabetes|May 28, 2017
Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca<sup>2+</sup>-Regulated GenesJennifer S Stancill, Jean-Philippe Cartailler, Hannah W Clayton, et al.
Diabetologia|February 25, 2026
The diabetes-associated K<sup>+</sup> channel TALK-2 controls human beta cell endoplasmic reticulum Ca<sup>2+</sup> handling, which promotes basal insulin release and limits glucose-stimulated insulin secretionJordyn R Dobson, Prasanna K Dadi, Matthew T Dickerson, et al.
Elife|May 3, 2024
The MODY-associated <i>KCNK16</i> L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adultsArya Y Nakhe, Prasanna K Dadi, Jinsun Kim, et al.
Biorxiv : the Preprint Server for Biology|August 7, 2023
The MODY-associated <i>KCNK16</i> L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adultsArya Y Nakhe, Prasanna K Dadi, Jinsun Kim, et al.
Cell Reports|January 11, 2024
TALK-1-mediated alterations of β-cell mitochondrial function and insulin secretion impair glucose homeostasis on a diabetogenic dietSarah M Graff, Arya Y Nakhe, Prasanna K Dadi, et al.
Diabetes, Obesity & Metabolism|May 13, 2022
Liraglutide increases islet Ca<sup>2+</sup> oscillation frequency and insulin secretion by activating hyperpolarization-activated cyclic nucleotide-gated channelsKarolina E Zaborska, Kelli L Jordan, Ariel S Thorson, et al.
Nature Communications|October 30, 2022
G<sub>i/o</sub> protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na<sup>+</sup>/K<sup>+</sup> ATPase activationMatthew T Dickerson, Prasanna K Dadi, Karolina E Zaborska, et al.
Pageof 3

Showing results (11-20 of 29) with videos related to

Sort By:
Pageof 3
Molecular Metabolism|February 7, 2018
TALK-1 reduces delta-cell endoplasmic reticulum and cytoplasmic calcium levels limiting somatostatin secretionNicholas C Vierra, Matthew T Dickerson, Kelli L Jordan, et al.
Molecular Metabolism|December 6, 2025
Glycolytic activation of β-cell Na<sup>+</sup>/K<sup>+</sup>-ATPases containing β1-subunits accelerates Na<sup>+</sup> extrusion, prolonging the duration of Ca<sup>2+</sup> oscillations but decreasing insulin secretionMatthew T Dickerson, Prasanna K Dadi, Reagan P McDevitt, et al.
American Journal of Physiology. Endocrinology and Metabolism|January 30, 2019
Glucose-mediated inhibition of calcium-activated potassium channels limits α-cell calcium influx and glucagon secretionMatthew T Dickerson, Prasanna K Dadi, Molly K Altman, et al.
Diabetes|May 28, 2017
Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca<sup>2+</sup>-Regulated GenesJennifer S Stancill, Jean-Philippe Cartailler, Hannah W Clayton, et al.
Diabetologia|February 25, 2026
The diabetes-associated K<sup>+</sup> channel TALK-2 controls human beta cell endoplasmic reticulum Ca<sup>2+</sup> handling, which promotes basal insulin release and limits glucose-stimulated insulin secretionJordyn R Dobson, Prasanna K Dadi, Matthew T Dickerson, et al.
Elife|May 3, 2024
The MODY-associated <i>KCNK16</i> L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adultsArya Y Nakhe, Prasanna K Dadi, Jinsun Kim, et al.
Biorxiv : the Preprint Server for Biology|August 7, 2023
The MODY-associated <i>KCNK16</i> L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adultsArya Y Nakhe, Prasanna K Dadi, Jinsun Kim, et al.
Cell Reports|January 11, 2024
TALK-1-mediated alterations of β-cell mitochondrial function and insulin secretion impair glucose homeostasis on a diabetogenic dietSarah M Graff, Arya Y Nakhe, Prasanna K Dadi, et al.
Diabetes, Obesity & Metabolism|May 13, 2022
Liraglutide increases islet Ca<sup>2+</sup> oscillation frequency and insulin secretion by activating hyperpolarization-activated cyclic nucleotide-gated channelsKarolina E Zaborska, Kelli L Jordan, Ariel S Thorson, et al.
Nature Communications|October 30, 2022
G<sub>i/o</sub> protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na<sup>+</sup>/K<sup>+</sup> ATPase activationMatthew T Dickerson, Prasanna K Dadi, Karolina E Zaborska, et al.
Pageof 3