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Cellular and Molecular Life Sciences : CMLS|June 6, 2022
Contact-dependent signaling triggers tumor-like proliferation of CCM3 knockout endothelial cells in co-culture with wild-type cellsMatthias Rath, Konrad Schwefel, Matteo Malinverno, et al.
European Journal of Medical Genetics|February 24, 2025
Oxford Nanopore long-read sequencing with CRISPR/Cas9-mediated target selection for accurate characterization of copy number variants in the LDLR geneRobin A Pilz, Dariush Skowronek, Loisa D Bonde, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|December 23, 2004
Non-heparan sulfate-binding interactions of endostatin/collagen XVIII in murine developmentNatalia Rychkova, Sonja Stahl, Sabine Gaetzner, et al.
Human Genetics|July 24, 2002
Centromeric association of chromosome 16- and 18-derived microchromosomesUte Felbor, Désirée Rutschow, Thomas Haaf, et al.
Journal of Negative Results in Biomedicine|June 2, 2006
VEGF receptors on PC12 cells mediate transient activation of ERK1/2 and Akt: comparison of nerve growth factor and vascular endothelial growth factorIngrid Berger, Sonja Stahl, Natalia Rychkova, et al.
Human Molecular Genetics|December 18, 2008
A two-hit mechanism causes cerebral cavernous malformations: complete inactivation of CCM1, CCM2 or CCM3 in affected endothelial cellsAxel Pagenstecher, Sonja Stahl, Ulrich Sure, et al.
Deutsches Arzteblatt International|March 18, 2020
Disorders Caused by Genetic MosaicismUte Moog, Ute Felbor, Cristina Has, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 10, 2020
Fibronectin rescues aberrant phenotype of endothelial cells lacking either CCM1, CCM2 or CCM3Konrad Schwefel, Stefanie Spiegler, Bettina C Kirchmaier, et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|August 24, 2005
Endostatin phenylalanines 31 and 34 define a receptor binding siteSonja Stahl, Sabine Gaetzner, Thomas D Mueller, et al.
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