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Blood|August 3, 2013
How T cells trigger the dissociation of the endothelial receptor phosphatase VE-PTP from VE-cadherinMatthias Vockel, Dietmar VestweberHuman Genetics|December 26, 2019
The X chromosome and male infertilityMatthias Vockel, Antoni Riera-Escamilla, Frank Tüttelmann, et al.Cell and Tissue Research|February 26, 2014
Phosphatases and kinases as regulators of the endothelial barrier functionVerena Küppers, Matthias Vockel, Astrid F Nottebaum, et al.Experimental Dermatology|July 16, 2010
Somatostatin regulates tight junction function and composition in human keratinocytesMatthias Vockel, Ute Breitenbach, Hans-Jürgen Kreienkamp, et al.Molecular & Cellular Proteomics : MCP|August 21, 2019
Vascular Endothelial Receptor Tyrosine Phosphatase: Identification of Novel Substrates Related to Junctions and a Ternary Complex with EPHB4 and TIE2Hannes C A Drexler, Matthias Vockel, Christian Polaschegg, et al.EMBO Reports|July 4, 2019
VE-PTP inhibition stabilizes endothelial junctions by activating FGD5Laura J Braun, Maren Zinnhardt, Matthias Vockel, et al.Plos One|May 19, 2011
Somatostatin inhibits cell migration and reduces cell counts of human keratinocytes and delays epidermal wound healing in an ex vivo wound modelMatthias Vockel, Simone Pollok, Ute Breitenbach, et al.The Journal of Experimental Medicine|December 9, 2015
Interfering with VE-PTP stabilizes endothelial junctions in vivo via Tie-2 in the absence of VE-cadherinMaike Frye, Martina Dierkes, Verena Küppers, et al.Nature Immunology|February 4, 2014
Leukocyte extravasation and vascular permeability are each controlled in vivo by different tyrosine residues of VE-cadherinFlorian Wessel, Mark Winderlich, Maren Holm, et al.FEBS Letters|December 17, 2008
Interaction of the human somatostatin receptor 3 with the multiple PDZ domain protein MUPP1 enables somatostatin to control permeability of epithelial tight junctionsChong Wee Liew, Matthias Vockel, Günter Glassmeier, et al.Pageof 2