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Journal of Neurophysiology|December 1, 2025
Cell-intrinsic mechanisms underlying spontaneous activity in the mouse visual cortical slice: implications for fragile X pathophysiologyMaxwell J Heinrich, Mark F BearBiorxiv : the Preprint Server for Biology|January 7, 2025
Non-ionotropic signaling through the NMDA receptor GluN2B carboxy terminal domain drives morphological plasticity of dendritic spines and reverses fragile X phenotypes in mouse hippocampusStephanie A Barnes, Aurore Thomazeau, Peter S B Finnie, et al.Cell Reports|February 21, 2025
Non-ionotropic signaling through the NMDA receptor GluN2B carboxy-terminal domain drives dendritic spine plasticity and reverses fragile X phenotypesStephanie A Barnes, Aurore Thomazeau, Peter S B Finnie, et al.Journal of Autoimmunity|March 4, 2019
Endogenous double-stranded Alu RNA elements stimulate IFN-responses in relapsing remitting multiple sclerosisMaxwell J Heinrich, Caroline A Purcell, Andrea J Pruijssers, et al.Biorxiv : the Preprint Server for Biology|April 1, 2025
A human electrophysiological biomarker of Fragile X Syndrome is shared in V1 of Fmr1 KO mice and caused by loss of FMRP in cortical excitatory neuronsSara S Kornfeld-Sylla, Cigdem Gelegen, Jordan E Norris, et al.Nature Communications|February 9, 2026
A human electrophysiological signature of Fragile X pathophysiology is shared in V1 of Fmr1-/y miceSara S Kornfeld-Sylla, Cigdem Gelegen, Jordan E Norris, et al.Biorxiv : the Preprint Server for Biology|April 10, 2026
FMR1 reduction alters cellular and circuit properties in human cortexAditi Singh, Saman Abbaspoor, Leeyup Chung, et al.Pageof 1