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Environment International|January 15, 2013
Role of genomic instability in arsenic-induced carcinogenicity. A reviewPritha Bhattacharjee, Mayukh Banerjee, Ashok K GiriToxicology and Applied Pharmacology|September 26, 2022
Zinc supplementation prevents mitotic accumulation in human keratinocyte cell lines upon environmentally relevant arsenic exposureMayukh Banerjee, Kavitha Yaddanapudi, J Christopher StatesJournal of Environmental Sciences (China)|December 24, 2016
Cellular arsenic transport pathways in mammalsBarbara A Roggenbeck, Mayukh Banerjee, Elaine M LeslieEnvironmental Toxicology and Pharmacology|June 28, 2022
Zinc supplementation prevents arsenic-induced dysregulation of ZRANB2 splice functionJonathan C Bastick, Mayukh Banerjee, J Christopher StatesArchives of Toxicology|February 5, 2022
Urolithin A attenuates arsenic-induced gut barrier dysfunctionSweta Ghosh, Mayukh Banerjee, Bodduluri Haribabu, et al.Mutation Research|July 22, 2008
Toxicogenomics of arsenic: classical ideas and recent advancesPritha Ghosh, Mayukh Banerjee, Ashok K Giri, et al.Toxicology and Applied Pharmacology|February 19, 2024
Chronic arsenic exposure induces malignant transformation of human HaCaT cells through both deterministic and stochastic changes in transcriptome expressionMayukh Banerjee, Sudhir Srivastava, Shesh N Rai, et al.Archives of Toxicology|January 14, 2026
Arsenic disrupts autophagosome-lysosome fusion in a zinc dependent manner across multiple human skin and lung cells linesRachel V Goff, Sidimohamed Elmoustapha, Shelia D Thomas, et al.International Journal of Cancer|April 5, 2008
DNA repair deficiency leads to susceptibility to develop arsenic-induced premalignant skin lesionsMayukh Banerjee, Nilendu Sarma, Rupanwita Biswas, et al.Toxicology and Applied Pharmacology|October 22, 2023
Clonal variability in chromosomal instability as a potential driver in the acquisition of tumorigenic phenotype in chronic arsenic-exposed and hsa-miR-186 overexpressing human keratinocytesAngeliki Lykoudi, Ana P Ferragut Cardoso, Sandra S Wise, et al.Pageof 5