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Biochemistry|May 6, 2022
C-Terminal Basic Region of Troponin T Alters the Ca<sup>2+</sup>-Dependent Changes in Troponin I InteractionsLi Zhu, Dylan Johnson, Joseph M ChalovichPlos One|December 11, 2012
Structural dynamics of troponin I during Ca2+-activation of cardiac thin filaments: a multi-site Förster resonance energy transfer studyHui Wang, Joseph M Chalovich, Gerard MarriottBiophysical Journal|September 4, 2004
Ca2+ and ionic strength dependencies of S1-ADP binding to actin-tropomyosin-troponin: regulatory implicationsBoris Gafurov, Yi-Der Chen, Joseph M ChalovichBiophysical Journal|September 18, 2007
Negative charges at protein kinase C sites of troponin I stabilize the inactive state of actinMohit C Mathur, Tomoyoshi Kobayashi, Joseph M ChalovichBiophysical Journal|July 31, 2018
Stepwise C-Terminal Truncation of Cardiac Troponin T Alters Function at Low and Saturating Ca<sup>2</sup>Dylan Johnson, C William Angus, Joseph M ChalovichJournal of Biomolecular Structure & Dynamics|July 22, 2010
Molecular dynamics studies on troponin (TnI-TnT-TnC) complexes: insight into the regulation of muscle contractionJayson F Varughese, Joseph M Chalovich, Yumin LiBiophysical Journal|March 18, 2009
Some cardiomyopathy-causing troponin I mutations stabilize a functional intermediate actin stateMohit C Mathur, Tomoyoshi Kobayashi, Joseph M ChalovichBiophysical Journal|July 19, 2002
Troponin-tropomyosin: an allosteric switch or a steric blocker?Andrea M Resetar, Jacqueline M Stephens, Joseph M ChalovichBiochemistry|October 3, 2013
Avian synaptopodin 2 (fesselin) stabilizes myosin filaments and actomyosin in the presence of ATPNathanial L Kingsbury, Randall H Renegar, Joseph M ChalovichBiochemical and Biophysical Research Communications|February 8, 2011
Several cardiomyopathy causing mutations on tropomyosin either destabilize the active state of actomyosin or alter the binding properties of tropomyosinMohit C Mathur, P Bryant Chase, Joseph M ChalovichPageof 5