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Mehmet M Altintas

Showing results (31-40 of 46) with videos related to

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The New England Journal of Medicine|November 6, 2015
Soluble Urokinase Receptor and Chronic Kidney DiseaseSalim S Hayek, Sanja Sever, Yi-An Ko, et al.
The Journal of Biological Chemistry|May 13, 2014
Reduction of proteinuria through podocyte alkalinizationMehmet M Altintas, Kumiko Moriwaki, Changli Wei, et al.
The Journal of Clinical Investigation|February 13, 2019
Nonimmune cell-derived ICOS ligand functions as a renoprotective αvβ3 integrin-selective antagonistKwi Hye Koh, Yanxia Cao, Steve Mangos, et al.
The Journal of Clinical Investigation|August 3, 2007
Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney diseaseSanja Sever, Mehmet M Altintas, Sharif R Nankoe, et al.
Nature Communications|May 3, 2022
Simultaneous stabilization of actin cytoskeleton in multiple nephron-specific cells protects the kidney from diverse injuryKamalika Mukherjee, Changkyu Gu, Agnieszka Collins, et al.
The Journal of Biological Chemistry|November 7, 2013
Transient receptor potential channel 6 (TRPC6) protects podocytes during complement-mediated glomerular diseaseAndreas D Kistler, Geetika Singh, Mehmet M Altintas, et al.
Kidney International|February 7, 2023
The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5Onur K Polat, Elena Isaeva, Yashwanth R Sudhini, et al.
Nature Medicine|December 18, 2007
Modification of kidney barrier function by the urokinase receptorChangli Wei, Clemens C Möller, Mehmet M Altintas, et al.
Nature Genetics|June 1, 2005
TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal functionJochen Reiser, Krishna R Polu, Clemens C Möller, et al.
The Journal of Biological Chemistry|December 4, 2016
Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1Ha Won Lee, Samia Q Khan, Shehryar Khaliqdina, et al.
Pageof 5

Showing results (31-40 of 46) with videos related to

Sort By:
Pageof 5
The New England Journal of Medicine|November 6, 2015
Soluble Urokinase Receptor and Chronic Kidney DiseaseSalim S Hayek, Sanja Sever, Yi-An Ko, et al.
The Journal of Biological Chemistry|May 13, 2014
Reduction of proteinuria through podocyte alkalinizationMehmet M Altintas, Kumiko Moriwaki, Changli Wei, et al.
The Journal of Clinical Investigation|February 13, 2019
Nonimmune cell-derived ICOS ligand functions as a renoprotective αvβ3 integrin-selective antagonistKwi Hye Koh, Yanxia Cao, Steve Mangos, et al.
The Journal of Clinical Investigation|August 3, 2007
Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney diseaseSanja Sever, Mehmet M Altintas, Sharif R Nankoe, et al.
Nature Communications|May 3, 2022
Simultaneous stabilization of actin cytoskeleton in multiple nephron-specific cells protects the kidney from diverse injuryKamalika Mukherjee, Changkyu Gu, Agnieszka Collins, et al.
The Journal of Biological Chemistry|November 7, 2013
Transient receptor potential channel 6 (TRPC6) protects podocytes during complement-mediated glomerular diseaseAndreas D Kistler, Geetika Singh, Mehmet M Altintas, et al.
Kidney International|February 7, 2023
The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5Onur K Polat, Elena Isaeva, Yashwanth R Sudhini, et al.
Nature Medicine|December 18, 2007
Modification of kidney barrier function by the urokinase receptorChangli Wei, Clemens C Möller, Mehmet M Altintas, et al.
Nature Genetics|June 1, 2005
TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal functionJochen Reiser, Krishna R Polu, Clemens C Möller, et al.
The Journal of Biological Chemistry|December 4, 2016
Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1Ha Won Lee, Samia Q Khan, Shehryar Khaliqdina, et al.
Pageof 5