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Biological Chemistry|June 21, 2018
Selective BH3-mimetics targeting BCL-2, BCL-XL or MCL-1 induce severe mitochondrial perturbationsKristina Henz, Aoula Al-Zebeeby, Marion Basoglu, et al.
British Journal of Haematology|January 26, 2013
Precision medicines for B-cell leukaemias and lymphomas; progress and potential pitfallsMartin J S Dyer, Meike Vogler, Jesvin Samuel, et al.
Journal of Molecular Medicine (Berlin, Germany)|August 10, 2021
Unleashing the power of NK cells in anticancer immunotherapyMeike Vogler, Senthan Shanmugalingam, Vinzenz Särchen, et al.
Cells|March 29, 2023
Characterization of BV6-Induced Sensitization to the NK Cell Killing of Pediatric Rhabdomyosarcoma SpheroidsVinzenz Särchen, Lisa Marie Reindl, Sara Wiedemann, et al.
British Journal of Haematology|October 6, 2025
The BH3-only protein NOXA is essential for apoptosis induction by BH3-mimetics targeting BCL2 or BCL-X<sub>L</sub> in DLBCLNahide Yildirim, Marius Anders, Victoria M Smith, et al.
NPJ Systems Biology and Applications|June 6, 2023
Computational modeling of DLBCL predicts response to BH3-mimeticsIelyaas Cloete, Victoria M Smith, Ross A Jackson, et al.
Haematologica|November 24, 2018
Targeting intermediary metabolism enhances the efficacy of BH3 mimetic therapy in hematologic malignanciesAoula Al-Zebeeby, Meike Vogler, Mateus Milani, et al.
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