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ACS Infectious Diseases|May 10, 2021
A Proteasome Mutation Sensitizes P. falciparum Cam3.II K13C580Y Parasites to DHA and OZ439Melissa R Rosenthal, Caroline L NgACS Infectious Diseases|April 24, 2020
Plasmodium falciparum Artemisinin Resistance: The Effect of Heme, Protein Damage, and Parasite Cell Stress ResponseMelissa R Rosenthal, Caroline L NgCell Reports Methods|August 3, 2023
High-content imaging as a tool to quantify and characterize malaria parasitesMelissa R Rosenthal, Caroline L NgMsphere|October 22, 2024
Enhanced cell stress response and protein degradation capacity underlie artemisinin resistance in Plasmodium falciparumMelissa R Rosenthal, Sukhithasri Vijayrajratnam, Tessa M Firestone, et al.Bioorganic & Medicinal Chemistry Letters|August 27, 2023
Identification of covalent fragment inhibitors for Plasmodium falciparum UCHL3 with anti-malarial efficacyRyan D Imhoff, Melissa R Rosenthal, Kutub Ashraf, et al.Biorxiv : the Preprint Server for Biology|September 2, 2025
Quinolines interfere with heme-mediated activation of artemisininsMelissa R Rosenthal, Daniel E GoldbergMethods in Molecular Biology (Clifton, N.J.)|July 4, 2019
Plasmodium falciparum In Vitro Drug Resistance Selections and Gene EditingCaroline L Ng, David A FidockExperimental Cell Research|May 1, 2010
TRAM1 is involved in disposal of ER membrane degradation substratesCaroline L Ng, Kristina Oresic, Domenico TortorellaThe Journal of Biological Chemistry|January 6, 2009
TRAM1 participates in human cytomegalovirus US2- and US11-mediated dislocation of an endoplasmic reticulum membrane glycoproteinKristina Oresic, Caroline L Ng, Domenico TortorellaTrends in Parasitology|July 10, 2017
Protein Degradation Systems as Antimalarial Therapeutic TargetsCaroline L Ng, David A Fidock, Matthew BogyoPageof 3