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Acta Physiologica (Oxford, England)|March 2, 2021
Zymogen-locked mutant prostasin (Prss8) leads to incomplete proteolytic activation of the epithelial sodium channel (ENaC) and severely compromises triamterene tolerance in miceDaniel Essigke, Alexandr V Ilyaskin, Matthias Wörn, et al.
Biotechnology and Applied Biochemistry|November 14, 2025
5-Methylcytosine Analysis of miRNAs in Minimal Change DiseaseHuiyi Zeng, Dandan Li, Yunyi Li, et al.
American Journal of Physiology. Renal Physiology|August 23, 2021
Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na+ channel in the mouse kidneyBernhard N Bohnert, Daniel Essigke, Andrea Janessa, et al.
Acta Physiologica (Oxford, England)|May 27, 2020
Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndromeMengyun Xiao, Bernhard N Bohnert, Hande Aypek, et al.
Disease Models & Mechanisms|August 23, 2021
Essential role of DNA-PKcs and plasminogen for the development of doxorubicin-induced glomerular injury in miceBernhard N Bohnert, Irene Gonzalez-Menendez, Thomas Dörffel, et al.
Kidney International|September 19, 2021
Proteinuric chronic kidney disease is associated with altered red blood cell lifespan, deformability and metabolismRosi Bissinger, Travis Nemkov, Angelo D'Alessandro, et al.
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