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Molecular Cell|November 7, 2020
ALS/FTLD-Linked Mutations in FUS Glycine Residues Cause Accelerated Gelation and Reduced Interactions with Wild-Type FUSKevin Rhine, Monika A Makurath, James Liu, et al.Biorxiv : the Preprint Server for Biology|November 24, 2025
RNA G-Quadruplexes Function as a Tunable Switch of FUS Phase SeparationJenny L Carey, Miyuki Hayashi, Emily Welebob, et al.Communications Biology|January 2, 2025
NLS-binding deficient Kapβ2 reduces neurotoxicity via selective interaction with C9orf72-ALS/FTD dipeptide repeatsKevin M Kim, Amandeep Girdhar, Maria E Cicardi, et al.The Journal of Biological Chemistry|October 21, 2009
N-terminal domains elicit formation of functional Pmel17 amyloid fibrilsBrenda Watt, Guillaume van Niel, Douglas M Fowler, et al.Molecular Cell|October 22, 2019
Loss of Dynamic RNA Interaction and Aberrant Phase Separation Induced by Two Distinct Types of ALS/FTD-Linked FUS MutationsAmirhossein Ghanbari Niaki, Jaya Sarkar, Xinyi Cai, et al.Nature Metabolism|July 8, 2026
Hypoxia rescues complex 1-associated disease caused by proteostatic defectsAnkur Garg, Brandon R Desousa, Raju Roy, et al.Biorxiv : the Preprint Server for Biology|July 14, 2025
Scouring the human Hsp70 network uncovers diverse chaperone safeguards buffering TDP-43 toxicityEdward M Barbieri, Miriam Linsenmeier, Katherine R Whiteman, et al.Nature Genetics|October 30, 2012
Inhibition of RNA lariat debranching enzyme suppresses TDP-43 toxicity in ALS disease modelsMaria Armakola, Matthew J Higgins, Matthew D Figley, et al.Molecular Biology of the Cell|December 12, 2019
The extent of Ssa1/Ssa2 Hsp70 chaperone involvement in nuclear protein quality control degradation varies with the substrateRamon D Jones, Charisma Enam, Rebeca Ibarra, et al.Cell Reports|December 28, 2020
Nuclear Import Receptors Directly Bind to Arginine-Rich Dipeptide Repeat Proteins and Suppress Their Pathological InteractionsSaskia Hutten, Sinem Usluer, Benjamin Bourgeois, et al.Pageof 21