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Current Medicinal Chemistry|June 17, 2017
The Role of Mitochondrial Dysfunction in the Progression of Alzheimer's DiseaseClaus Desler, Meryl S Lillenes, Tone Tønjum, et al.
Mechanisms of Ageing and Development|September 3, 2011
DNA base excision repair gene polymorphisms modulate human cognitive performance and decline during normal life spanMeryl S Lillenes, Thomas Espeseth, Mari Støen, et al.
Mechanisms of Ageing and Development|October 15, 2013
Transient OGG1, APE1, PARP1 and Polβ expression in an Alzheimer's disease mouse modelMeryl S Lillenes, Mari Støen, Marta Gómez-Muñoz, et al.
Neuroscience Letters|March 1, 2017
Mitochondrial transcription factor A (TFAM) rs1937 and AP endonuclease 1 (APE1) rs1130409 alleles are associated with reduced cognitive performanceMeryl S Lillenes, Mari Støen, Clara-Cecilie Günther, et al.
Neuroscience Letters|February 24, 2018
Expression of nucleotide excision repair in Alzheimer's disease is higher in brain tissue than in bloodHelge Leander B Jensen, Meryl S Lillenes, Alberto Rabano, et al.
Molecular Brain|May 29, 2016
Altered DNA base excision repair profile in brain tissue and blood in Alzheimer's diseaseMeryl S Lillenes, Alberto Rabano, Mari Støen, et al.
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