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Mi Bai

Showing results (1-10 of 39) with videos related to

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Roux'S Archives of Developmental Biology : the Official Organ of the EDBO|March 18, 2017
A quantitive evaluation of gap junctions and their morphological alteration during differentiation of amphibian notochord cellsZeng Mi-Bai, Zhou Mei-Yun
American Journal of Physiology. Cell Physiology|December 9, 2020
The emerging role of MEIS1 in cell proliferation and differentiationMingzhu Jiang, Shuang Xu, Mi Bai, et al.
Pediatric Nephrology (Berlin, Germany)|June 7, 2023
Long-term treatment of chronic kidney disease patients with anemia using hypoxia-inducible factor prolyl hydroxylase inhibitors: potential concernsJia He, Zhanjun Jia, Aihua Zhang, et al.
In Vitro Cellular & Developmental Biology. Animal|March 20, 2026
A preliminary study on the mechanism of stromal interaction molecule 1 (STIM1) involvement in Adriamycin-induced podocyte injuryLi Miao, Mi Bai, Songming Huang, et al.
Kidney Diseases (Basel, Switzerland)|October 30, 2023
From Physiology to Pathology: The Role of Mitochondria in Acute Kidney Injuries and Chronic Kidney DiseasesLingge Zhang, Mengqiu Miao, Xinyue Xu, et al.
American Journal of Physiology. Renal Physiology|October 19, 2020
Mitochondrial dysfunction and the AKI-to-CKD transitionMingzhu Jiang, Mi Bai, Juan Lei, et al.
Oncotarget|March 5, 2016
PGC-1α overexpression protects against aldosterone-induced podocyte depletion: role of mitochondriaMin Zhao, Yanggang Yuan, Mi Bai, et al.
Kidney Diseases (Basel, Switzerland)|July 13, 2018
Angiotensin II Stimulates the NLRP3 Inflammasome to Induce Podocyte Injury and Mitochondrial DysfunctionMin Zhao, Mi Bai, Guixia Ding, et al.
Advanced Science (Weinheim, Baden-Wurttemberg, Germany)|March 17, 2025
HNF3α Targets Nckap1l and Promotes Renal Fibrosis Following Ischemia-Reperfusion InjuryLing Hou, Yan Guo, Shuang Xu, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|February 10, 2023
CDC20 inhibition alleviates fibrotic response of renal tubular epithelial cells and fibroblasts by regulating nuclear translocation of β-cateninJia He, Shuang Xu, Mingzhu Jiang, et al.
Pageof 4

Showing results (1-10 of 39) with videos related to

Sort By:
Pageof 4
Roux'S Archives of Developmental Biology : the Official Organ of the EDBO|March 18, 2017
A quantitive evaluation of gap junctions and their morphological alteration during differentiation of amphibian notochord cellsZeng Mi-Bai, Zhou Mei-Yun
American Journal of Physiology. Cell Physiology|December 9, 2020
The emerging role of MEIS1 in cell proliferation and differentiationMingzhu Jiang, Shuang Xu, Mi Bai, et al.
Pediatric Nephrology (Berlin, Germany)|June 7, 2023
Long-term treatment of chronic kidney disease patients with anemia using hypoxia-inducible factor prolyl hydroxylase inhibitors: potential concernsJia He, Zhanjun Jia, Aihua Zhang, et al.
In Vitro Cellular & Developmental Biology. Animal|March 20, 2026
A preliminary study on the mechanism of stromal interaction molecule 1 (STIM1) involvement in Adriamycin-induced podocyte injuryLi Miao, Mi Bai, Songming Huang, et al.
Kidney Diseases (Basel, Switzerland)|October 30, 2023
From Physiology to Pathology: The Role of Mitochondria in Acute Kidney Injuries and Chronic Kidney DiseasesLingge Zhang, Mengqiu Miao, Xinyue Xu, et al.
American Journal of Physiology. Renal Physiology|October 19, 2020
Mitochondrial dysfunction and the AKI-to-CKD transitionMingzhu Jiang, Mi Bai, Juan Lei, et al.
Oncotarget|March 5, 2016
PGC-1α overexpression protects against aldosterone-induced podocyte depletion: role of mitochondriaMin Zhao, Yanggang Yuan, Mi Bai, et al.
Kidney Diseases (Basel, Switzerland)|July 13, 2018
Angiotensin II Stimulates the NLRP3 Inflammasome to Induce Podocyte Injury and Mitochondrial DysfunctionMin Zhao, Mi Bai, Guixia Ding, et al.
Advanced Science (Weinheim, Baden-Wurttemberg, Germany)|March 17, 2025
HNF3α Targets Nckap1l and Promotes Renal Fibrosis Following Ischemia-Reperfusion InjuryLing Hou, Yan Guo, Shuang Xu, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|February 10, 2023
CDC20 inhibition alleviates fibrotic response of renal tubular epithelial cells and fibroblasts by regulating nuclear translocation of β-cateninJia He, Shuang Xu, Mingzhu Jiang, et al.
Pageof 4