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Cell|January 17, 2015
Leptin and insulin act on POMC neurons to promote the browning of white fatGarron T Dodd, Stephanie Decherf, Kim Loh, et al.Elife|September 20, 2018
Insulin regulates POMC neuronal plasticity to control glucose metabolismGarron T Dodd, Natalie J Michael, Robert S Lee-Young, et al.The Journal of Clinical Investigation|September 10, 2014
The arcuate nucleus mediates GLP-1 receptor agonist liraglutide-dependent weight lossAnna Secher, Jacob Jelsing, Arian F Baquero, et al.Journal of Medicinal Chemistry|December 12, 2018
Celastrol Promotes Weight Loss in Diet-Induced Obesity by Inhibiting the Protein Tyrosine Phosphatases PTP1B and TCPTP in the HypothalamusEleni Kyriakou, Stefanie Schmidt, Garron T Dodd, et al.Neuron|January 21, 2006
Leptin directly activates SF1 neurons in the VMH, and this action by leptin is required for normal body-weight homeostasisHarveen Dhillon, Jeffrey M Zigman, Chianping Ye, et al.Proceedings of the National Academy of Sciences of the United States of America|August 4, 2010
Synaptic input organization of the melanocortin system predicts diet-induced hypothalamic reactive gliosis and obesityTamas L Horvath, Beatrix Sarman, Cristina García-Cáceres, et al.Molecular Metabolism|March 8, 2021
The melanocortin pathway and energy homeostasis: From discovery to obesity therapyGiles S H Yeo, Daniela Herrera Moro Chao, Anna-Maria Siegert, et al.Cell Metabolism|October 18, 2011
Elevated hypothalamic TCPTP in obesity contributes to cellular leptin resistanceKim Loh, Atsushi Fukushima, Xinmei Zhang, et al.Nature|February 25, 2015
Hypothalamic POMC neurons promote cannabinoid-induced feedingMarco Koch, Luis Varela, Jae Geun Kim, et al.Diabetes|August 31, 2018
Celastrol-Induced Weight Loss Is Driven by Hypophagia and Independent From UCP1Katrin Pfuhlmann, Sonja C Schriever, Peter Baumann, et al.Pageof 10