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Plos Genetics|March 22, 2018
Impaired proteoglycan glycosylation, elevated TGF-β signaling, and abnormal osteoblast differentiation as the basis for bone fragility in a mouse model for gerodermia osteodysplasticaWing Lee Chan, Magdalena Steiner, Tomasz Witkos, et al.Cell Metabolism|June 4, 2010
Glucocorticoids suppress bone formation by attenuating osteoblast differentiation via the monomeric glucocorticoid receptorAlexander Rauch, Sebastian Seitz, Ulrike Baschant, et al.Brain : a Journal of Neurology|January 25, 2014
Sensory neuropathy with bone destruction due to a mutation in the membrane-shaping atlastin GTPase 3Uwe Kornak, Inès Mademan, Marte Schinke, et al.Science Translational Medicine|May 6, 2026
Extracellular traps from myeloid cells as therapeutic targets in traumatic heterotopic ossificationShan Jiang, Gesine Eis-Janzyk, Ruben Augustin, et al.The Journal of Cell Biology|March 16, 2011
Control of bone formation by the serpentine receptor Frizzled-9Joachim Albers, Jochen Schulze, F Timo Beil, et al.Bone Research|February 23, 2024
Piezo1 expression in chondrocytes controls endochondral ossification and osteoarthritis developmentLaura J Brylka, Assil-Ramin Alimy, Miriam E A Tschaffon-Müller, et al.Nature Medicine|May 19, 2009
Impaired gastric acidification negatively affects calcium homeostasis and bone massThorsten Schinke, Arndt F Schilling, Anke Baranowsky, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|August 4, 2018
The Lysosomal Protein Arylsulfatase B Is a Key Enzyme Involved in Skeletal TurnoverSandra Pohl, Alexandra Angermann, Anke Jeschke, et al.Nature Communications|October 22, 2014
Calcitonin controls bone formation by inhibiting the release of sphingosine 1-phosphate from osteoclastsJohannes Keller, Philip Catala-Lehnen, Antje K Huebner, et al.American Journal of Human Genetics|March 19, 2013
Mutations in WNT1 cause different forms of bone fragilityKatharina Keupp, Filippo Beleggia, Hülya Kayserili, et al.Pageof 36