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Heart Rhythm|July 13, 2010
Optimizing catecholaminergic polymorphic ventricular tachycardia therapy in calsequestrin-mutant miceGuy Katz, Assad Khoury, Efrat Kurtzwald, et al.
EMBO Molecular Medicine|February 22, 2017
SK4 K<sup>+</sup> channels are therapeutic targets for the treatment of cardiac arrhythmiasShiraz Haron-Khun, David Weisbrod, Hanna Bueno, et al.
International Journal of Molecular Sciences|September 9, 2022
Bioenergetic and Metabolic Impairments in Induced Pluripotent Stem Cell-Derived Cardiomyocytes Generated from Duchenne Muscular Dystrophy PatientsLubna Willi, Ifat Abramovich, Jonatan Fernandez-Garcia, et al.
The Journal of Clinical Investigation|February 6, 2002
Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathyMichael Arad, D Woodrow Benson, Antonio R Perez-Atayde, et al.
ESC Heart Failure|April 30, 2022
Hypokinetic hypertrophic cardiomyopathy: clinical phenotype, genetics, and prognosisYishay Wasserstrum, José M Larrañaga-Moreira, Cristina Martinez-Veira, et al.
FEBS Letters|April 7, 2005
Functional role of AMP-activated protein kinase in the heart during exerciseNicolas Musi, Michael F Hirshman, Michael Arad, et al.
Heart Rhythm|April 29, 2014
Alpha blockade potentiates CPVT therapy in calsequestrin-mutant miceEfrat Kurtzwald-Josefson, Edith Hochhauser, Katia Bogachenko, et al.
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