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Michael Brownlee

Showing results (1-10 of 88) with videos related to

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Diabetes|May 28, 2005
The pathobiology of diabetic complications: a unifying mechanismMichael Brownlee
Diabetes|January 23, 2016
Response to Comment on Giacco et al. GLP-1 Cleavage Product Reverses Persistent ROS Generation After Transient Hyperglycemia by Disrupting an ROS-Generating Feedback Loop. Diabetes 2015;64:3273-3284Michael Brownlee
Nature Medicine|November 8, 2007
Preventing kidney cell suicideMichael Brownlee
The Journal of Clinical Investigation|December 18, 2003
A radical explanation for glucose-induced beta cell dysfunctionMichael Brownlee
Cell Metabolism|May 14, 2013
Hyperglycemia-stimulated myelopoiesis causes impaired regression of atherosclerosis in type 1 diabetesMichael Brownlee
Diabetes|October 17, 2009
Hyperglycemia-induced reactive oxygen species increase expression of the receptor for advanced glycation end products (RAGE) and RAGE ligandsDachun Yao, Michael Brownlee
Circulation Research|October 30, 2010
Oxidative stress and diabetic complicationsFerdinando Giacco, Michael Brownlee
Annals of Internal Medicine|October 5, 2018
Hemoglobin A1c Targets for Glycemic Control With Pharmacologic TherapyJoel Zonszein, Michael Brownlee
JAMA|June 10, 2010
Beyond hemoglobin A1c--need for additional markers of risk for diabetic microvascular complicationsIrl B Hirsch, Michael Brownlee
Diabetes Care|December 29, 2006
The effect of glucose variability on the risk of microvascular complications in type 1 diabetesIrl B Hirsch, Michael Brownlee
Pageof 9

Showing results (1-10 of 88) with videos related to

Sort By:
Pageof 9
Diabetes|May 28, 2005
The pathobiology of diabetic complications: a unifying mechanismMichael Brownlee
Diabetes|January 23, 2016
Response to Comment on Giacco et al. GLP-1 Cleavage Product Reverses Persistent ROS Generation After Transient Hyperglycemia by Disrupting an ROS-Generating Feedback Loop. Diabetes 2015;64:3273-3284Michael Brownlee
Nature Medicine|November 8, 2007
Preventing kidney cell suicideMichael Brownlee
The Journal of Clinical Investigation|December 18, 2003
A radical explanation for glucose-induced beta cell dysfunctionMichael Brownlee
Cell Metabolism|May 14, 2013
Hyperglycemia-stimulated myelopoiesis causes impaired regression of atherosclerosis in type 1 diabetesMichael Brownlee
Diabetes|October 17, 2009
Hyperglycemia-induced reactive oxygen species increase expression of the receptor for advanced glycation end products (RAGE) and RAGE ligandsDachun Yao, Michael Brownlee
Circulation Research|October 30, 2010
Oxidative stress and diabetic complicationsFerdinando Giacco, Michael Brownlee
Annals of Internal Medicine|October 5, 2018
Hemoglobin A1c Targets for Glycemic Control With Pharmacologic TherapyJoel Zonszein, Michael Brownlee
JAMA|June 10, 2010
Beyond hemoglobin A1c--need for additional markers of risk for diabetic microvascular complicationsIrl B Hirsch, Michael Brownlee
Diabetes Care|December 29, 2006
The effect of glucose variability on the risk of microvascular complications in type 1 diabetesIrl B Hirsch, Michael Brownlee
Pageof 9