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Allergy|March 23, 2023
Anti-CD3 inhibits circulatory and tissue-resident memory CD4 T cells that drive asthma exacerbations in miceGurupreet S Sethi, Donald T Gracias, Rinkesh K Gupta, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 3, 2017
HVEM Imprints Memory Potential on Effector CD8 T Cells Required for Protective Mucosal ImmunityPritesh Desai, Georges Abboud, Jessica Stanfield, et al.
American Journal of Transplantation : Official Journal of the American Society of Transplantation and the American Society of Transplant Surgeons|December 22, 2017
The TWEAK/Fn14 pathway is required for calcineurin inhibitor toxicity of the kidneysMeike Claus, Rana Herro, Dennis Wolf, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 7, 2007
CD134 Costimulation Couples the CD137 Pathway to Induce Production of Supereffector CD8 T Cells That Become IL-7 DependentSeung-Joo Lee, Robert J Rossi, Sun-Kyeong Lee, et al.
American Journal of Clinical Dermatology|January 18, 2024
OX40 in the Pathogenesis of Atopic Dermatitis-A New Therapeutic TargetMichael Croft, Ehsanollah Esfandiari, Camilla Chong, et al.
European Journal of Immunology|April 26, 2013
Transgenic expression of survivin compensates for OX40-deficiency in driving Th2 development and allergic inflammationFengyang Lei, Jianyong Song, Rizwanul Haque, et al.
Science Immunology|November 19, 2021
TWEAK functions with TNF and IL-17 on keratinocytes and is a potential target for psoriasis therapyRinkesh K Gupta, Donald T Gracias, Daniela Salgado Figueroa, et al.
The Journal of Allergy and Clinical Immunology|June 24, 2015
Rhinovirus infection interferes with induction of tolerance to aeroantigens through OX40 ligand, thymic stromal lymphopoietin, and IL-33Amit K Mehta, Wei Duan, Astrid M Doerner, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 7, 2007
OX40 costimulation promotes persistence of cytomegalovirus-specific CD8 T Cells: A CD4-dependent mechanismIan R Humphreys, Andrea Loewendorf, Carl de Trez, et al.
The Journal of Clinical Investigation|July 28, 2007
SLAT regulates Th1 and Th2 inflammatory responses by controlling Ca2+/NFAT signalingStéphane Bécart, Céline Charvet, Ann J Canonigo Balancio, et al.
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