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Journal of Immunology (Baltimore, Md. : 1950)|May 22, 2007
Vaccinia virus-specific CD4+ T cell responses target a set of antigens largely distinct from those targeted by CD8+ T cell responsesMagdalini Moutaftsi, Huynh-Hoa Bui, Bjoern Peters, et al.Journal of Immunology (Baltimore, Md. : 1950)|September 22, 2020
TL1A Promotes Lung Tissue Fibrosis and Airway RemodelingRana Herro, Haruka Miki, Gurupreet S Sethi, et al.Gastroenterology|July 27, 2020
Increased Production of LIGHT by T Cells in Eosinophilic Esophagitis Promotes Differentiation of Esophageal Fibroblasts Toward an Inflammatory PhenotypeMario C Manresa, Austin W T Chiang, Richard C Kurten, et al.Cell Reports|April 9, 2020
TLR9 Sensing of Self-DNA Controls Cell-Mediated Immunity to Listeria Infection via Rapid Conversion of Conventional CD4<sup>+</sup> T Cells to T<sub>reg</sub>Joseph S Dolina, Joey Lee, Ryan Q Griswold, et al.Plos Pathogens|January 18, 2024
Late-rising CD4 T cells resolve mouse cytomegalovirus persistent replication in the salivary glandSimon Brunel, Gaelle Picarda, Ankan Gupta, et al.Science Signaling|October 3, 2013
The TNF family member 4-1BBL sustains inflammation by interacting with TLR signaling components during late-phase activationJianhui Ma, Bo-Ram Bang, Jiawei Lu, et al.Blood|August 27, 2005
Costimulation of mast cells by 4-1BB, a member of the tumor necrosis factor receptor superfamily, with the high-affinity IgE receptorHajime Nishimoto, Seung-Woo Lee, Hong Hong, et al.The Journal of Allergy and Clinical Immunology|December 14, 2020
The deubiquitinase CYLD controls protective immunity against helminth infection by regulation of Treg cell plasticityJee H Lee, Le Zou, Runqing Yang, et al.Blood|July 20, 2014
Accumulation of 4-1BBL+ B cells in the elderly induces the generation of granzyme-B+ CD8+ T cells with potential antitumor activityCatalina Lee-Chang, Monica Bodogai, Kanako Moritoh, et al.Journal of Immunology (Baltimore, Md. : 1950)|November 19, 2014
OX40- and CD27-mediated costimulation synergizes with anti-PD-L1 blockade by forcing exhausted CD8+ T cells to exit quiescenceSarah Buchan, Teresa Manzo, Barry Flutter, et al.Pageof 18