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Journal of Molecular and Cellular Cardiology|August 30, 2005
Greater propensity of diabetic myocardium for oxidative stress after myocardial infarction is associated with the development of heart failureHolly M Smith, Milton Hamblin, Michael F Hill
Circulation Research|October 30, 2012
Neuregulin in cardiovascular development and diseaseOghenerukevwe Odiete, Michael F Hill, Douglas B Sawyer
Molecular and Cellular Biochemistry|July 2, 2004
Antioxidant and oxidative stress changes in experimental cor pulmonaleFiroozeh Farahmand, Michael F Hill, Pawan K Singal
Cardiovascular Pathology : the Official Journal of the Society for Cardiovascular Pathology|April 10, 2013
Augmented cardiac formation of oxidatively-induced carbonylated proteins accompanies the increased functional severity of post-myocardial infarction heart failure in the setting of type 1 diabetes mellitusKathleen E Dennis, Salisha Hill, Kristie L Rose, et al.
Experimental and Clinical Cardiology|July 31, 2009
Reduction in oxidative stress and modulation of heart failure subsequent to myocardial infarction in ratsMichael F Hill, Vince P Palace, Kuljeet Kaur, et al.
Journal of Molecular and Cellular Cardiology|February 27, 2007
Alterations in the diabetic myocardial proteome coupled with increased myocardial oxidative stress underlies diabetic cardiomyopathyMilton Hamblin, David B Friedman, Salisha Hill, et al.
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