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Oxidative Medicine and Cellular Longevity|June 14, 2019
The Mitochondrial Permeability Transition in Mitochondrial DisordersJustina Šileikytė, Michael ForteJournal of Bioenergetics and Biomembranes|September 17, 2005
Genetic dissection of the permeability transition poreMichael Forte, Paolo BernardiBiochimica Et Biophysica Acta|March 1, 2016
Shutting down the pore: The search for small molecule inhibitors of the mitochondrial permeability transitionJustina Šileikytė, Michael ForteThe Biochemical Journal|June 5, 2003
Response of yeast to the regulated expression of proteins in the Bcl-2 familyPeter Polcic, Michael ForteNovartis Foundation Symposium|December 14, 2007
The mitochondrial permeability transition porePaolo Bernardi, Michael ForteFrontiers in Physiology|July 27, 2012
Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challengesKimmy Su, Dennis Bourdette, Michael ForteFrontiers in Physiology|July 31, 2013
Mitochondrial dysfunction and neurodegeneration in multiple sclerosisKimmy Su, Dennis Bourdette, Michael ForteThe European Journal of Neuroscience|July 5, 2011
Mitochondrial calcium and its regulation in neurodegeneration induced by oxidative stressAnna G Barsukova, Dennis Bourdette, Michael ForteThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 31, 2012
Focal increases of axoplasmic Ca2+, aggregation of sodium-calcium exchanger, N-type Ca2+ channel, and actin define the sites of spheroids in axons undergoing oxidative stressAnna G Barsukova, Michael Forte, Dennis BourdettePhysiological Reviews|August 14, 2015
The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in PathophysiologyPaolo Bernardi, Andrea Rasola, Michael Forte, et al.Pageof 4