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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 22, 2016
Short Fibrils Constitute the Major Species of Seed-Competent Tau in the Brains of Mice Transgenic for Human P301S TauSamuel J Jackson, Caroline Kerridge, Jane Cooper, et al.
European Spine Journal : Official Publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society|May 26, 2023
Predictors of poor outcome following lumbar spinal fusion surgery: a prospective observational study to derive two clinical prediction rules using British Spine Registry dataAlison B Rushton, Feroz Jadhakhan, Martin L Verra, et al.
Neurobiology of Disease|March 16, 2002
A presenilin 1 mutation associated with familial frontotemporal dementia inhibits gamma-secretase cleavage of APP and notchZareen Amtul, Patrick A Lewis, Sian Piper, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 14, 2006
An inhibitor of tau hyperphosphorylation prevents severe motor impairments in tau transgenic miceSylvie Le Corre, Hans W Klafki, Nikolaus Plesnila, et al.
Human Molecular Genetics|October 16, 2003
Fine mapping of the alpha-T catenin gene to a quantitative trait locus on chromosome 10 in late-onset Alzheimer's disease pedigreesNilüfer Ertekin-Taner, James Ronald, Hideaki Asahara, et al.
The Journal of Clinical Investigation|February 17, 2007
The high-affinity HSP90-CHIP complex recognizes and selectively degrades phosphorylated tau client proteinsChad A Dickey, Adeela Kamal, Karen Lundgren, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|May 8, 2016
Tau: From research to clinical developmentDavid M Holtzman, Maria C Carrillo, James A Hendrix, et al.
Brain : a Journal of Neurology|January 13, 2006
A family with tau-negative frontotemporal dementia and neuronal intranuclear inclusions linked to chromosome 17Ian R Mackenzie, Matthew Baker, Gemma West, et al.
Neurobiology of Aging|July 18, 2021
A clinical, molecular genetics and pathological study of a FTDP-17 family with a heterozygous splicing variant c.823-10G>T at the intron 9/exon 10 of the MAPT geneDiana A Olszewska, Conor Fearon, Christopher McGuigan, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 30, 2006
Deletion of the ubiquitin ligase CHIP leads to the accumulation, but not the aggregation, of both endogenous phospho- and caspase-3-cleaved tau speciesChad A Dickey, Mei Yue, Wen-Lang Lin, et al.
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