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Michael I Sadowski

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Protein Science : a Publication of the Protein Society|November 22, 2011
Protein topology from predicted residue contactsWilliam R Taylor, David T Jones, Michael I Sadowski
Current Opinion in Structural Biology|May 18, 2013
Prediction of contacts from correlated sequence substitutionsWilliam R Taylor, Russell S Hamilton, Michael I Sadowski
Proteins|December 18, 2009
De novo backbone scaffolds for protein designJames T MacDonald, Katarzyna Maksimiak, Michael I Sadowski, et al.
Computational Biology and Chemistry|June 28, 2011
Exploring the limits of fold discrimination by structural alignment: a large scale benchmark using decoys of known foldSiv Midtun Hollup, Michael I Sadowski, Inge Jonassen, et al.
Plos One|June 14, 2012
Analytic markovian rates for generalized protein structure evolutionIvan Coluzza, James T MacDonald, Michael I Sadowski, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 9, 2004
Genome-wide analysis of repressor element 1 silencing transcription factor/neuron-restrictive silencing factor (REST/NRSF) target genesAlexander W Bruce, Ian J Donaldson, Ian C Wood, et al.
Human Molecular Genetics|September 23, 2010
FOXRED1, encoding an FAD-dependent oxidoreductase complex-I-specific molecular chaperone, is mutated in infantile-onset mitochondrial encephalopathyElisa Fassone, Andrew J Duncan, Jan-Willem Taanman, et al.
Human Molecular Genetics|May 30, 2015
FOXRED1, encoding an FAD-dependent oxidoreductase complex-I-specific molecular chaperone, is mutated in infantile-onset mitochondrial encephalopathyElisa Fassone, Andrew J Duncan, Jan-Willem Taanman, et al.
American Journal of Human Genetics|April 21, 2009
A nonsense mutation in COQ9 causes autosomal-recessive neonatal-onset primary coenzyme Q10 deficiency: a potentially treatable form of mitochondrial diseaseAndrew J Duncan, Maria Bitner-Glindzicz, Brigitte Meunier, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 21, 2007
The implications of alternative splicing in the ENCODE protein complementMichael L Tress, Pier Luigi Martelli, Adam Frankish, et al.
Pageof 2

Showing results (11-20 of 20) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 20 results.
Protein Science : a Publication of the Protein Society|November 22, 2011
Protein topology from predicted residue contactsWilliam R Taylor, David T Jones, Michael I Sadowski
Current Opinion in Structural Biology|May 18, 2013
Prediction of contacts from correlated sequence substitutionsWilliam R Taylor, Russell S Hamilton, Michael I Sadowski
Proteins|December 18, 2009
De novo backbone scaffolds for protein designJames T MacDonald, Katarzyna Maksimiak, Michael I Sadowski, et al.
Computational Biology and Chemistry|June 28, 2011
Exploring the limits of fold discrimination by structural alignment: a large scale benchmark using decoys of known foldSiv Midtun Hollup, Michael I Sadowski, Inge Jonassen, et al.
Plos One|June 14, 2012
Analytic markovian rates for generalized protein structure evolutionIvan Coluzza, James T MacDonald, Michael I Sadowski, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 9, 2004
Genome-wide analysis of repressor element 1 silencing transcription factor/neuron-restrictive silencing factor (REST/NRSF) target genesAlexander W Bruce, Ian J Donaldson, Ian C Wood, et al.
Human Molecular Genetics|September 23, 2010
FOXRED1, encoding an FAD-dependent oxidoreductase complex-I-specific molecular chaperone, is mutated in infantile-onset mitochondrial encephalopathyElisa Fassone, Andrew J Duncan, Jan-Willem Taanman, et al.
Human Molecular Genetics|May 30, 2015
FOXRED1, encoding an FAD-dependent oxidoreductase complex-I-specific molecular chaperone, is mutated in infantile-onset mitochondrial encephalopathyElisa Fassone, Andrew J Duncan, Jan-Willem Taanman, et al.
American Journal of Human Genetics|April 21, 2009
A nonsense mutation in COQ9 causes autosomal-recessive neonatal-onset primary coenzyme Q10 deficiency: a potentially treatable form of mitochondrial diseaseAndrew J Duncan, Maria Bitner-Glindzicz, Brigitte Meunier, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 21, 2007
The implications of alternative splicing in the ENCODE protein complementMichael L Tress, Pier Luigi Martelli, Adam Frankish, et al.
Pageof 2