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Journal of the American College of Cardiology|May 11, 2019
Myofibroblast Phenotype and Reversibility of Fibrosis in Patients With End-Stage Heart FailureChandan K Nagaraju, Emma L Robinson, Mouna Abdesselem, et al.
The Journal of Biological Chemistry|May 30, 2002
Mechanisms underlying the neuronal calcium sensor-1-evoked enhancement of exocytosis in PC12 cellsSchuichi Koizumi, Patrizia Rosa, Gary B Willars, et al.
Basic Research in Cardiology|November 15, 2022
InsP<sub>3</sub>R-RyR Ca<sup>2+</sup> channel crosstalk facilitates arrhythmias in the failing human ventricleXin Jin, Matthew Amoni, Guillaume Gilbert, et al.
Matrix Biology Plus|December 11, 2023
Temporal expression and spatial distribution of the proteoglycan versican during cardiac fibrosis developmentAthiramol Sasi, Andreas Romaine, Pugazendhi Murugan Erusappan, et al.
Advanced Healthcare Materials|February 12, 2026
Patient-Derived 3D Bioprinted Cardiac Organoid Constructs Reveal Key Pathological Features of Duchenne Muscular DystrophyVittoria Marini, Margalida Campaner Socias, Andreas Dimopoulos, et al.
Neurochemistry International|July 19, 2011
Non-immortalized human neural stem (NS) cells as a scalable platform for cellular assaysLilian Hook, Joaquim Vives, Norma Fulton, et al.
Cells|February 25, 2022
MSK-Mediated Phosphorylation of Histone H3 Ser28 Couples MAPK Signalling with Early Gene Induction and Cardiac HypertrophyEmma L Robinson, Faye M Drawnel, Saher Mehdi, et al.
Cell Death & Disease|September 8, 2023
Intracellular BAPTA directly inhibits PFKFB3, thereby impeding mTORC1-driven Mcl-1 translation and killing MCL-1-addicted cancer cellsFlore Sneyers, Martijn Kerkhofs, Femke Speelman-Rooms, et al.
Cardiovascular Research|February 16, 2026
Sodium-myo-inositol cotransporter-1, SMIT1, promotes cardiac hypertrophy and fibrosis induced by pressure overload in miceAlice Marino, Julien Cumps, Laura Guilbert, et al.
Biochimica Et Biophysica Acta. Molecular Cell Research|January 13, 2022
A non-canonical role for pyruvate kinase M2 as a functional modulator of Ca<sup>2+</sup> signalling through IP<sub>3</sub> receptorsAndrew R Lavik, Karen S McColl, Fernanda O Lemos, et al.
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