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Neuropharmacology|September 12, 2007
A selective, non-peptide caspase-1 inhibitor, VRT-018858, markedly reduces brain damage induced by transient ischemia in the ratJerard Ross, David Brough, Rosemary M Gibson, et al.Journal of Neuroinflammation|December 31, 2011
Interleukin-1α expression precedes IL-1β after ischemic brain injury and is localised to areas of focal neuronal loss and penumbral tissuesNadia M Luheshi, Krisztina J Kovács, Gloria Lopez-Castejon, et al.The Journal of Biological Chemistry|May 3, 2014
Release of interleukin-1α or interleukin-1β depends on mechanism of cell deathHazel England, Holly R Summersgill, Michelle E Edye, et al.Journal of Neuroinflammation|August 26, 2016
Decreased haemodynamic response and decoupling of cortical gamma-band activity and tissue oxygen perfusion after striatal interleukin-1 injectionNatasha Bray, Fiona E Burrows, Myles Jones, et al.Plos One|March 11, 2011
Relative roles of the cellular and humoral responses in the Drosophila host defense against three gram-positive bacterial infectionsNadine T Nehme, Jessica Quintin, Ju Hyun Cho, et al.Plos Pathogens|May 21, 2019
Innate immune memory through TLR2 and NOD2 contributes to the control of Leptospira interrogans infectionIgnacio Santecchia, Frédérique Vernel-Pauillac, Orhan Rasid, et al.Brain Communications|November 2, 2020
Anti-inflammatories in Alzheimer's disease-potential therapy or spurious correlate?Jack Rivers-Auty, Alison E Mather, Ruth Peters, et al.The Journal of Biological Chemistry|November 14, 2001
Priming of macrophages with lipopolysaccharide potentiates P2X7-mediated cell death via a caspase-1-dependent mechanism, independently of cytokine productionRosalind A Le Feuvre, David Brough, Yoichiro Iwakura, et al.Frontiers in Neuroscience|January 31, 2014
Surgical manipulation compromises leukocyte mobilization responses and inflammation after experimental cerebral ischemia in miceAdam Denes, Jesus M Pradillo, Caroline Drake, et al.Neurobiology of Aging|March 18, 2014
High-fat diet-induced memory impairment in triple-transgenic Alzheimer's disease (3xTgAD) mice is independent of changes in amyloid and tau pathologyElysse M Knight, Isaura V A Martins, Sarah Gümüsgöz, et al.Pageof 32