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Biorxiv : the Preprint Server for Biology|November 24, 2025
Axon Regeneration and Functional Recovery after Spinal Cord Injury is Enhanced by Allele-Specific ApoE Neuronal Action through LRP8Ramakrishnan Kannan, Xingxing Wang, LaShae Nicholson, et al.
Biorxiv : the Preprint Server for Biology|August 8, 2025
mGluR4-Npdc1 complex mediates α-synuclein fibril-induced neurodegenerationAzucena Perez-Canamas, Mingming Chen, Leire Almandoz-Gil, et al.
Molecular Imaging and Biology|January 12, 2022
Translational PET Imaging of Spinal Cord Injury with the Serotonin Transporter Tracer [11C]AFMHanyi Fang, Samantha Rossano, Xingxing Wang, et al.
The Journal of Biological Chemistry|May 29, 2015
Prion-Protein-interacting Amyloid-β Oligomers of High Molecular Weight Are Tightly Correlated with Memory Impairment in Multiple Alzheimer Mouse ModelsMikhail A Kostylev, Adam C Kaufman, Haakon B Nygaard, et al.
Nature Communications|February 16, 2024
Reduced progranulin increases tau and α-synuclein inclusions and alters mouse tauopathy phenotypes via glucocerebrosidaseHideyuki Takahashi, Sanaea Bhagwagar, Sarah H Nies, et al.
Nature Methods|November 6, 2019
Simultaneous mesoscopic and two-photon imaging of neuronal activity in cortical circuitsDaniel Barson, Ali S Hamodi, Xilin Shen, et al.
Neuron|November 25, 2010
Sortilin-mediated endocytosis determines levels of the frontotemporal dementia protein, progranulinFenghua Hu, Thihan Padukkavidana, Christian B Vægter, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 3, 2006
Alzheimer precursor protein interaction with the Nogo-66 receptor reduces amyloid-beta plaque depositionJames H Park, David A Gimbel, Tadzia GrandPre, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 13, 2006
Identification of a receptor necessary for Nogo-B stimulated chemotaxis and morphogenesis of endothelial cellsRobert Qing Miao, Yuan Gao, Kenneth D Harrison, et al.
Acta Neuropathologica Communications|July 3, 2020
Fyn kinase inhibition reduces protein aggregation, increases synapse density and improves memory in transgenic and traumatic TauopathySi Jie Tang, Arman Fesharaki-Zadeh, Hideyuki Takahashi, et al.
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