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Archives of Neurology|November 11, 2009
Cellular prion protein mediates the toxicity of beta-amyloid oligomers: implications for Alzheimer diseaseHaakon B Nygaard, Stephen M StrittmatterAdvances in Pharmacology (San Diego, Calif.)|February 8, 2018
Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5A Harrison Brody, Stephen M StrittmatterJournal of Molecular Medicine (Berlin, Germany)|December 5, 2009
Beta-amyloid oligomers and cellular prion protein in Alzheimer's diseaseErik C Gunther, Stephen M StrittmatterTrends in Neurosciences|April 12, 2003
The Nogo-66 receptor: focusing myelin inhibition of axon regenerationAaron W McGee, Stephen M StrittmatterGenome Biology|January 8, 2008
The reticulons: a family of proteins with diverse functionsYvonne S Yang, Stephen M StrittmatterExperimental Neurology|April 9, 2008
Functional MRI and other non-invasive imaging technologies: providing visual biomarkers for spinal cord structure and function after injuryNoam Y Harel, Stephen M StrittmatterCurrent Alzheimer Research|January 29, 2008
Nogo receptor interacts with brain APP and Abeta to reduce pathologic changes in Alzheimer's transgenic miceJames H Park, Stephen M StrittmatterPrion|September 19, 2012
Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer diseaseJi Won Um, Stephen M StrittmatterNature Reviews. Neuroscience|July 22, 2006
Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury?Noam Y Harel, Stephen M StrittmatterResults and Problems in Cell Differentiation|July 8, 2009
Axon regeneration in the peripheral and central nervous systemsEric A Huebner, Stephen M StrittmatterPageof 25