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Neuron|October 27, 2004
Nogo-66 receptor prevents raphespinal and rubrospinal axon regeneration and limits functional recovery from spinal cord injuryJi-Eun Kim, Betty P Liu, James H Park, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 22, 2002
Truncated soluble Nogo receptor binds Nogo-66 and blocks inhibition of axon growth by myelinAlyson E Fournier, Graham C Gould, Betty P Liu, et al.Progress in Brain Research|November 21, 2002
Nogo and the Nogo-66 receptorAlyson E Fournier, Tadzia GrandPré, Graham Gould, et al.Biorxiv : the Preprint Server for Biology|September 18, 2025
Insulin resistance alters cortical inhibitory neurons and microglia to exacerbate Alzheimer's knock-in mouse phenotypesLaShae Nicholson, Si Jie Tang, Tejaswini Karra, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 27, 2019
Plexina2 and CRMP2 Signaling Complex Is Activated by Nogo-A-Liganded Ngr1 to Restrict Corticospinal Axon Sprouting after TraumaYuichi Sekine, Percy T Algarate, William B J Cafferty, et al.Biomarkers : Biochemical Indicators of Exposure, Response, and Susceptibility to Chemicals|June 25, 2009
Serum Nogo-A levels are not elevated in amyotrophic lateral sclerosis patientsNoam Y Harel, Merit E Cudkowicz, Robert H Brown, et al.Brain Research|September 20, 2014
Progressive retinal degeneration and accumulation of autofluorescent lipopigments in Progranulin deficient miceBrian P Hafler, Zoe A Klein, Z Jimmy Zhou, et al.Biorxiv : the Preprint Server for Biology|September 25, 2023
Lysosomal TMEM106B interacts with galactosylceramidase to regulate myelin lipid metabolismHideyuki Takahashi, Azucena Perez-Canamas, Hongping Ye, et al.Molecular and Cellular Neurosciences|July 29, 2014
Lysosome size, motility and stress response regulated by fronto-temporal dementia modifier TMEM106BMassimiliano Stagi, Zoe A Klein, Travis J Gould, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 30, 2015
Plasticity of intact rubral projections mediates spontaneous recovery of function after corticospinal tract injuryChad S Siegel, Kathren L Fink, Stephen M Strittmatter, et al.Pageof 25