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Molecular Pharmacology|August 24, 2004
Evidence for a novel K(+) channel modulated by alpha(1A)-adrenoceptors in cardiac myocytesStéphanie C M Choisy, Jules C Hancox, Lesley A Arberry, et al.
The Journal of Biological Chemistry|August 27, 2011
The inhibitory effect of phospholemman on the sodium pump requires its palmitoylationLindsay B Tulloch, Jacqueline Howie, Krzysztof J Wypijewski, et al.
American Journal of Physiology. Cell Physiology|April 3, 2009
FXYD1 phosphorylation in vitro and in adult rat cardiac myocytes: threonine 69 is a novel substrate for protein kinase CWilliam Fuller, Jacqueline Howie, Linda M McLatchie, et al.
Cardiovascular Research|November 2, 2020
Off-target effects of sodium-glucose co-transporter 2 blockers: empagliflozin does not inhibit Na+/H+ exchanger-1 or lower [Na+]i in the heartYu Jin Chung, Kyung Chan Park, Sergiy Tokar, et al.
Cardiovascular Research|April 4, 2017
Restitution slope is principally determined by steady-state action potential durationMichael J Shattock, Kyung Chan Park, Hsiang-Yu Yang, et al.
Cardiovascular Research|December 29, 2004
Serine 68 phosphorylation of phospholemman: acute isoform-specific activation of cardiac Na/K ATPaseBenjamin d Z Silverman, William Fuller, Philip Eaton, et al.
Journal of Molecular and Cellular Cardiology|April 25, 2013
Nitric oxide regulates cardiac intracellular Na⁺ and Ca²⁺ by modulating Na/K ATPase via PKCε and phospholemman-dependent mechanismDavor Pavlovic, Andrew R Hall, Erika J Kennington, et al.
Biochemical and Biophysical Research Communications|June 5, 2019
Ablation of Toll-like receptor 9 attenuates myocardial ischemia/reperfusion injury in miceRika Kitazume-Taneike, Manabu Taneike, Shigemiki Omiya, et al.
American Journal of Physiology. Heart and Circulatory Physiology|June 15, 2010
Phospholemman Ser69 phosphorylation contributes to sildenafil-induced cardioprotection against reperfusion injuryMelanie Madhani, Andrew R Hall, Friederike Cuello, et al.
The Journal of Physiology|August 23, 2008
Maternal dietary supplementation with saturated, but not monounsaturated or polyunsaturated fatty acids, leads to tissue-specific inhibition of offspring Na+,K+-ATPaseJames A Armitage, Sanjana Gupta, Caroline Wood, et al.
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