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The European Journal of Neuroscience|July 12, 2013
Striatal dopamine transmission is reduced after chronic nicotine with a decrease in α6-nicotinic receptor control in nucleus accumbensRichard Exley, Michael A Clements, Henrike Hartung, et al.
Journal of Lipid Research|December 25, 2012
Diacylglycerol kinase inhibitor R59022 attenuates conjugated linoleic acid-mediated inflammation in human adipocytesKristina Martinez, Shruthi Shyamasundar, Arion Kennedy, et al.
The Journal of Pharmacology and Experimental Therapeutics|April 19, 2002
Characterization of nicotinic agonist-induced [(3)H]dopamine release from synaptosomes prepared from four mouse brain regionsSharon R Grady, Karen L Murphy, Jian Cao, et al.
Molecular Pharmacology|March 8, 2007
Pharmacology of alpha-conotoxin MII-sensitive subtypes of nicotinic acetylcholine receptors isolated by breeding of null mutant miceOuti Salminen, Jennifer A Drapeau, J Michael McIntosh, et al.
Biochemical Pharmacology|May 26, 2011
Characterizing functional α6β2 nicotinic acetylcholine receptors in vitro: mutant β2 subunits improve membrane expression, and fluorescent proteins reveal responsive cellsCheng Xiao, Rahul Srinivasan, Ryan M Drenan, et al.
Movement Disorders : Official Journal of the Movement Disorder Society|November 18, 2015
α7 nicotinic receptor agonists reduce levodopa-induced dyskinesias with severe nigrostriatal damageDanhui Zhang, Matthew McGregor, Tanuja Bordia, et al.
International Journal of Biological Macromolecules|April 12, 2026
Single amino acid substitutions modulate the function of α9α10 nicotinic acetylcholine receptorXuan Zhou, Xiu Jiang, Shengrong Pei, et al.
Plos One|April 15, 2014
Monkey adrenal chromaffin cells express α6β4* nicotinic acetylcholine receptorsAlicia Hernández-Vivanco, Arik J Hone, Mick L Scadden, et al.
The Journal of Pharmacology and Experimental Therapeutics|April 21, 2007
Chronic nicotine differentially regulates alpha6- and beta3-containing nicotinic cholinergic receptors in rat brainDavid C Perry, Danyan Mao, Allison B Gold, et al.
The Journal of Biological Chemistry|February 26, 2015
Key residues in the nicotinic acetylcholine receptor β2 subunit contribute to α-conotoxin LvIA bindingDongting Zhangsun, Xiaopeng Zhu, Yong Wu, et al.
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