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The Journal of Biological Chemistry
|
September 16, 2003
Superoxide activates uncoupling proteins by generating carbon-centered radicals and initiating lipid peroxidation: studies using a mitochondria-targeted spin trap derived from alpha-phenyl-N-tert-butylnitrone
Michael P Murphy, Karim S Echtay, Frances H Blaikie, et al.
Nature Communications
|
November 9, 2023
Failure to apply standard limit-of-detection or limit-of-quantitation criteria to specialized pro-resolving mediator analysis incorrectly characterizes their presence in biological samples
Valerie B O'Donnell, Nils H Schebb, Ginger L Milne, et al.
Scientific Data
|
December 3, 2022
A multi-omics longitudinal aging dataset in primary human fibroblasts with mitochondrial perturbations
Gabriel Sturm, Anna S Monzel, Kalpita R Karan, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism
|
November 1, 2016
Focally perfused succinate potentiates brain metabolism in head injury patients
Ibrahim Jalloh, Adel Helmy, Duncan J Howe, et al.
Redox Biology
|
September 1, 2020
Targeting succinate dehydrogenase with malonate ester prodrugs decreases renal ischemia reperfusion injury
Timothy E Beach, Hiran A Prag, Laura Pala, et al.
The American Journal of Pathology
|
September 25, 2018
Placental Adaptation to Early-Onset Hypoxic Pregnancy and Mitochondria-Targeted Antioxidant Therapy in a Rodent Model
Anna M Nuzzo, Emily J Camm, Amanda N Sferruzzi-Perri, et al.
Nature Metabolism
|
February 19, 2025
Pro-inflammatory macrophages produce mitochondria-derived superoxide by reverse electron transport at complex I that regulates IL-1β release during NLRP3 inflammasome activation
Alva M Casey, Dylan G Ryan, Hiran A Prag, et al.
Cancers
|
March 25, 2022
MitoQ Inhibits Human Breast Cancer Cell Migration, Invasion and Clonogenicity
Tania Capeloa, Joanna Krzystyniak, Donatienne d'Hose, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
May 6, 2025
Loss of RET-ROS at complex I induces diastolic dysfunction in mice that is reversed by aerobic exercise
Ana Vujic, Amy Koo, Guillaume Bidault, et al.
Circulation
|
July 12, 2013
Mitochondrial DNA damage can promote atherosclerosis independently of reactive oxygen species through effects on smooth muscle cells and monocytes and correlates with higher-risk plaques in humans
Emma Yu, Patrick A Calvert, John R Mercer, et al.
Page
of 57
Search research articles
Search
Showing results (471-480 of 561) with videos related to
Sort By:
Page
of 57
The Journal of Biological Chemistry
|
September 16, 2003
Superoxide activates uncoupling proteins by generating carbon-centered radicals and initiating lipid peroxidation: studies using a mitochondria-targeted spin trap derived from alpha-phenyl-N-tert-butylnitrone
Michael P Murphy, Karim S Echtay, Frances H Blaikie, et al.
Nature Communications
|
November 9, 2023
Failure to apply standard limit-of-detection or limit-of-quantitation criteria to specialized pro-resolving mediator analysis incorrectly characterizes their presence in biological samples
Valerie B O'Donnell, Nils H Schebb, Ginger L Milne, et al.
Scientific Data
|
December 3, 2022
A multi-omics longitudinal aging dataset in primary human fibroblasts with mitochondrial perturbations
Gabriel Sturm, Anna S Monzel, Kalpita R Karan, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism
|
November 1, 2016
Focally perfused succinate potentiates brain metabolism in head injury patients
Ibrahim Jalloh, Adel Helmy, Duncan J Howe, et al.
Redox Biology
|
September 1, 2020
Targeting succinate dehydrogenase with malonate ester prodrugs decreases renal ischemia reperfusion injury
Timothy E Beach, Hiran A Prag, Laura Pala, et al.
The American Journal of Pathology
|
September 25, 2018
Placental Adaptation to Early-Onset Hypoxic Pregnancy and Mitochondria-Targeted Antioxidant Therapy in a Rodent Model
Anna M Nuzzo, Emily J Camm, Amanda N Sferruzzi-Perri, et al.
Nature Metabolism
|
February 19, 2025
Pro-inflammatory macrophages produce mitochondria-derived superoxide by reverse electron transport at complex I that regulates IL-1β release during NLRP3 inflammasome activation
Alva M Casey, Dylan G Ryan, Hiran A Prag, et al.
Cancers
|
March 25, 2022
MitoQ Inhibits Human Breast Cancer Cell Migration, Invasion and Clonogenicity
Tania Capeloa, Joanna Krzystyniak, Donatienne d'Hose, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
May 6, 2025
Loss of RET-ROS at complex I induces diastolic dysfunction in mice that is reversed by aerobic exercise
Ana Vujic, Amy Koo, Guillaume Bidault, et al.
Circulation
|
July 12, 2013
Mitochondrial DNA damage can promote atherosclerosis independently of reactive oxygen species through effects on smooth muscle cells and monocytes and correlates with higher-risk plaques in humans
Emma Yu, Patrick A Calvert, John R Mercer, et al.
Page
of 57