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Nature Genetics|October 4, 2005
Second-generation shRNA libraries covering the mouse and human genomesJose M Silva, Mamie Z Li, Ken Chang, et al.Nature Medicine|December 19, 2018
Tumor-derived IFN triggers chronic pathway agonism and sensitivity to ADAR lossHuayang Liu, Javad Golji, Lauren K Brodeur, et al.Nature Medicine|April 8, 2015
Studying clonal dynamics in response to cancer therapy using high-complexity barcodingHyo-eun C Bhang, David A Ruddy, Viveksagar Krishnamurthy Radhakrishna, et al.Cancer Discovery|June 5, 2016
CRISPR Screens Provide a Comprehensive Assessment of Cancer Vulnerabilities but Generate False-Positive Hits for Highly Amplified Genomic RegionsDiana M Munoz, Pamela J Cassiani, Li Li, et al.Elife|February 2, 2017
KEAP1 loss modulates sensitivity to kinase targeted therapy in lung cancerElsa B Krall, Belinda Wang, Diana M Munoz, et al.The Journal of Clinical Investigation|December 15, 2023
Rational design of a SOCS1-edited tumor-infiltrating lymphocyte therapy using CRISPR/Cas9 screensMichael R Schlabach, Sharon Lin, Zachary R Collester, et al.Science (New York, N.Y.)|December 14, 2011
A SUMOylation-dependent transcriptional subprogram is required for Myc-driven tumorigenesisJessica D Kessler, Kristopher T Kahle, Tingting Sun, et al.Science (New York, N.Y.)|February 26, 2016
Disordered methionine metabolism in MTAP/CDKN2A-deleted cancers leads to dependence on PRMT5Konstantinos J Mavrakis, E Robert McDonald, Michael R Schlabach, et al.Cell|July 29, 2017
Project DRIVE: A Compendium of Cancer Dependencies and Synthetic Lethal Relationships Uncovered by Large-Scale, Deep RNAi ScreeningE Robert McDonald, Antoine de Weck, Michael R Schlabach, et al.Pageof 2