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American Journal of Physiology. Lung Cellular and Molecular Physiology|October 12, 2002
Hypoxic but not anoxic stabilization of HIF-1alpha requires mitochondrial reactive oxygen speciesClara Schroedl, David S McClintock, G R Scott Budinger, et al.
Cell Reports|April 13, 2016
The Mitochondrial Respiratory Chain Is Required for Organismal Adaptation to HypoxiaRobert B Hamanaka, Samuel E Weinberg, Colleen R Reczek, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|May 11, 2002
Mitochondrial ROS initiate phosphorylation of p38 MAP kinase during hypoxia in cardiomyocytesAndre Kulisz, Ningfang Chen, Navdeep S Chandel, et al.
Molecular and Cellular Biology|October 31, 2007
Loss of the SdhB, but Not the SdhA, subunit of complex II triggers reactive oxygen species-dependent hypoxia-inducible factor activation and tumorigenesisRobert D Guzy, Bhumika Sharma, Eric Bell, et al.
Translational Research : the Journal of Laboratory and Clinical Medicine|October 29, 2017
Reactive oxygen species as signaling molecules in the development of lung fibrosisFrancisco J Gonzalez-Gonzalez, Navdeep S Chandel, Manu Jain, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 13, 2008
PTEN regulates p300-dependent hypoxia-inducible factor 1 transcriptional activity through Forkhead transcription factor 3a (FOXO3a)Brooke M Emerling, Frank Weinberg, Juinn-Lin Liu, et al.
The Journal of Clinical Investigation|December 3, 2013
Targeting SOD1 reduces experimental non–small-cell lung cancerAndrea Glasauer, Laura A Sena, Lauren P Diebold, et al.
Molecular and Cellular Biology|June 15, 2007
Mitochondrial reactive oxygen species trigger hypoxia-inducible factor-dependent extension of the replicative life span during hypoxiaEric L Bell, Tatyana A Klimova, James Eisenbart, et al.
Blood and Lymphatic Cancer : Targets and Therapy|February 4, 2020
The expanding role of venetoclax in chronic lymphocytic leukemia and small lymphocytic lymphomaMichael Schieber, Shuo Ma
The Journal of Biological Chemistry|November 20, 2003
Reactive oxygen species are required for hyperoxia-induced Bax activation and cell death in alveolar epithelial cellsLeonard J Buccellato, May Tso, Ozkan I Akinci, et al.
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