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Michael T Ryan

Showing results (121-130 of 136) with videos related to

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Nature Communications|July 11, 2020
Publisher Correction: Metabolic characteristics of CD8<sup>+</sup> T cell subsets in young and aged individuals are not predictive of functionalityKylie M Quinn, Tabinda Hussain, Felix Kraus, et al.
Plos Genetics|January 4, 2014
Mutations in the UQCC1-interacting protein, UQCC2, cause human complex III deficiency associated with perturbed cytochrome b protein expressionElena J Tucker, Bas F J Wanschers, Radek Szklarczyk, et al.
The EMBO Journal|July 21, 2022
AIFM1 is a component of the mitochondrial disulfide relay that drives complex I assembly through efficient import of NDUFS5Silja Lucia Salscheider, Sarah Gerlich, Alfredo Cabrera-Orefice, et al.
Nature Communications|April 13, 2023
High ploidy large cytoplasmic megakaryocytes are hematopoietic stem cells regulators and essential for platelet productionShen Y Heazlewood, Tanveer Ahmad, Benjamin Cao, et al.
Cell Metabolism|November 20, 2021
Quantitative high-confidence human mitochondrial proteome and its dynamics in cellular contextMarcel Morgenstern, Christian D Peikert, Philipp Lübbert, et al.
Nature Communications|May 12, 2021
Nicotinamide riboside attenuates age-associated metabolic and functional changes in hematopoietic stem cellsXuan Sun, Benjamin Cao, Marina Naval-Sanchez, et al.
Science (New York, N.Y.)|February 24, 2018
BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosisKate McArthur, Lachlan W Whitehead, John M Heddleston, et al.
EMBO Molecular Medicine|September 12, 2018
<i>OXA1L</i> mutations cause mitochondrial encephalopathy and a combined oxidative phosphorylation defectKyle Thompson, Nicole Mai, Monika Oláhová, et al.
Nature Communications|November 28, 2018
VDAC2 enables BAX to mediate apoptosis and limit tumor developmentHui San Chin, Mark X Li, Iris K L Tan, et al.
American Journal of Human Genetics|September 25, 2018
Bi-allelic Mutations in NDUFA6 Establish Its Role in Early-Onset Isolated Mitochondrial Complex I DeficiencyCharlotte L Alston, Juliana Heidler, Marris G Dibley, et al.
Pageof 14

Showing results (121-130 of 136) with videos related to

Sort By:
Pageof 14
Nature Communications|July 11, 2020
Publisher Correction: Metabolic characteristics of CD8<sup>+</sup> T cell subsets in young and aged individuals are not predictive of functionalityKylie M Quinn, Tabinda Hussain, Felix Kraus, et al.
Plos Genetics|January 4, 2014
Mutations in the UQCC1-interacting protein, UQCC2, cause human complex III deficiency associated with perturbed cytochrome b protein expressionElena J Tucker, Bas F J Wanschers, Radek Szklarczyk, et al.
The EMBO Journal|July 21, 2022
AIFM1 is a component of the mitochondrial disulfide relay that drives complex I assembly through efficient import of NDUFS5Silja Lucia Salscheider, Sarah Gerlich, Alfredo Cabrera-Orefice, et al.
Nature Communications|April 13, 2023
High ploidy large cytoplasmic megakaryocytes are hematopoietic stem cells regulators and essential for platelet productionShen Y Heazlewood, Tanveer Ahmad, Benjamin Cao, et al.
Cell Metabolism|November 20, 2021
Quantitative high-confidence human mitochondrial proteome and its dynamics in cellular contextMarcel Morgenstern, Christian D Peikert, Philipp Lübbert, et al.
Nature Communications|May 12, 2021
Nicotinamide riboside attenuates age-associated metabolic and functional changes in hematopoietic stem cellsXuan Sun, Benjamin Cao, Marina Naval-Sanchez, et al.
Science (New York, N.Y.)|February 24, 2018
BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosisKate McArthur, Lachlan W Whitehead, John M Heddleston, et al.
EMBO Molecular Medicine|September 12, 2018
<i>OXA1L</i> mutations cause mitochondrial encephalopathy and a combined oxidative phosphorylation defectKyle Thompson, Nicole Mai, Monika Oláhová, et al.
Nature Communications|November 28, 2018
VDAC2 enables BAX to mediate apoptosis and limit tumor developmentHui San Chin, Mark X Li, Iris K L Tan, et al.
American Journal of Human Genetics|September 25, 2018
Bi-allelic Mutations in NDUFA6 Establish Its Role in Early-Onset Isolated Mitochondrial Complex I DeficiencyCharlotte L Alston, Juliana Heidler, Marris G Dibley, et al.
Pageof 14