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Michael Wagenbach

Showing results (1-10 of 30) with videos related to

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The Journal of Cell Biology|November 26, 2002
K-loop insertion restores microtubule depolymerizing activity of a "neckless" MCAK mutantYulia Ovechkina, Michael Wagenbach, Linda Wordeman
The Journal of Cell Biology|November 28, 2007
MCAK facilitates chromosome movement by promoting kinetochore microtubule turnoverLinda Wordeman, Michael Wagenbach, George von Dassow
The Journal of Cell Biology|November 13, 2008
A kinesin-13 mutant catalytically depolymerizes microtubules in ADPMichael Wagenbach, Sarah Domnitz, Linda Wordeman, et al.
Molecular Biology of the Cell|February 26, 2016
Divergent microtubule assembly rates after short- versus long-term loss of end-modulating kinesinsLinda Wordeman, Justin Decarreau, Juan Jesus Vicente, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 29, 2025
The Kinesin Motor Kif9 Disrupts Primary Cilia Length by Mispositioning Centriolar SatellitesJuan Jesus Vicente, Jacob Weiss, Michael Wagenbach, et al.
The Journal of Cell Biology|April 12, 2012
MCAK activity at microtubule tips regulates spindle microtubule length to promote robust kinetochore attachmentSarah B Domnitz, Michael Wagenbach, Justin Decarreau, et al.
Current Protocols|December 28, 2023
Production of CRISPR-Cas9 Transgenic Cell Lines for Knocksideways StudiesMichael Wagenbach, Juan Jesus Vicente, Wren Wagenbach, et al.
Nature Structural & Molecular Biology|December 8, 2009
Catalysis of the microtubule on-rate is the major parameter regulating the depolymerase activity of MCAKJeremy R Cooper, Michael Wagenbach, Charles L Asbury, et al.
Developmental Cell|May 19, 2012
Kif18A and chromokinesins confine centromere movements via microtubule growth suppression and spatial control of kinetochore tensionJason Stumpff, Michael Wagenbach, Andrew Franck, et al.
FEBS Letters|August 12, 2004
MCAK, a Kin I kinesin, increases the catastrophe frequency of steady-state HeLa cell microtubules in an ATP-dependent manner in vitroCori N Newton, Michael Wagenbach, Yulia Ovechkina, et al.
Pageof 3

Showing results (1-10 of 30) with videos related to

Sort By:
Pageof 3
The Journal of Cell Biology|November 26, 2002
K-loop insertion restores microtubule depolymerizing activity of a "neckless" MCAK mutantYulia Ovechkina, Michael Wagenbach, Linda Wordeman
The Journal of Cell Biology|November 28, 2007
MCAK facilitates chromosome movement by promoting kinetochore microtubule turnoverLinda Wordeman, Michael Wagenbach, George von Dassow
The Journal of Cell Biology|November 13, 2008
A kinesin-13 mutant catalytically depolymerizes microtubules in ADPMichael Wagenbach, Sarah Domnitz, Linda Wordeman, et al.
Molecular Biology of the Cell|February 26, 2016
Divergent microtubule assembly rates after short- versus long-term loss of end-modulating kinesinsLinda Wordeman, Justin Decarreau, Juan Jesus Vicente, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 29, 2025
The Kinesin Motor Kif9 Disrupts Primary Cilia Length by Mispositioning Centriolar SatellitesJuan Jesus Vicente, Jacob Weiss, Michael Wagenbach, et al.
The Journal of Cell Biology|April 12, 2012
MCAK activity at microtubule tips regulates spindle microtubule length to promote robust kinetochore attachmentSarah B Domnitz, Michael Wagenbach, Justin Decarreau, et al.
Current Protocols|December 28, 2023
Production of CRISPR-Cas9 Transgenic Cell Lines for Knocksideways StudiesMichael Wagenbach, Juan Jesus Vicente, Wren Wagenbach, et al.
Nature Structural & Molecular Biology|December 8, 2009
Catalysis of the microtubule on-rate is the major parameter regulating the depolymerase activity of MCAKJeremy R Cooper, Michael Wagenbach, Charles L Asbury, et al.
Developmental Cell|May 19, 2012
Kif18A and chromokinesins confine centromere movements via microtubule growth suppression and spatial control of kinetochore tensionJason Stumpff, Michael Wagenbach, Andrew Franck, et al.
FEBS Letters|August 12, 2004
MCAK, a Kin I kinesin, increases the catastrophe frequency of steady-state HeLa cell microtubules in an ATP-dependent manner in vitroCori N Newton, Michael Wagenbach, Yulia Ovechkina, et al.
Pageof 3