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Michaela Fenckova

Showing results (11-20 of 23) with videos related to

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Research Square|February 12, 2024
Pre-Pulse Inhibition of an escape response in adult fruit fly, <i>Drosophila melanogaster</i>Erika Viragh, Lenke Asztalos, Michaela Fenckova, et al.
Biological Psychiatry|July 6, 2019
Habituation Learning Is a Widely Affected Mechanism in Drosophila Models of Intellectual Disability and Autism Spectrum DisordersMichaela Fenckova, Laura E R Blok, Lenke Asztalos, et al.
Translational Psychiatry|January 8, 2026
Pre-Pulse Inhibition of an escape response in adult fruit fly, Drosophila melanogasterErika Viragh, Lenke Asztalos, Michaela Fenckova, et al.
Plos Genetics|May 12, 2016
BOD1 Is Required for Cognitive Function in Humans and DrosophilaSahar Esmaeeli-Nieh, Michaela Fenckova, Iain M Porter, et al.
Plos One|February 13, 2019
Conserved regulation of neurodevelopmental processes and behavior by FoxP in DrosophilaAnna Castells-Nobau, Ilse Eidhof, Michaela Fenckova, et al.
Plos Genetics|October 26, 2017
Functional convergence of histone methyltransferases EHMT1 and KMT2C involved in intellectual disability and autism spectrum disorderTom S Koemans, Tjitske Kleefstra, Melissa C Chubak, et al.
Human Molecular Genetics|April 12, 2013
CEP89 is required for mitochondrial metabolism and neuronal function in man and flyBregje W M van Bon, Merel A W Oortveld, Leo G Nijtmans, et al.
American Journal of Human Genetics|June 26, 2012
Disruption of an EHMT1-associated chromatin-modification module causes intellectual disabilityTjitske Kleefstra, Jamie M Kramer, Kornelia Neveling, et al.
Journal of Medical Genetics|May 7, 2013
GATAD2B loss-of-function mutations cause a recognisable syndrome with intellectual disability and are associated with learning deficits and synaptic undergrowth in DrosophilaMarjolein H Willemsen, Bonnie Nijhof, Michaela Fenckova, et al.
European Journal of Human Genetics : EJHG|January 14, 2016
De novo loss-of-function mutations in WAC cause a recognizable intellectual disability syndrome and learning deficits in DrosophilaDorien Lugtenberg, Margot R F Reijnders, Michaela Fenckova, et al.
Pageof 3

Showing results (11-20 of 23) with videos related to

Sort By:
Pageof 3
Research Square|February 12, 2024
Pre-Pulse Inhibition of an escape response in adult fruit fly, <i>Drosophila melanogaster</i>Erika Viragh, Lenke Asztalos, Michaela Fenckova, et al.
Biological Psychiatry|July 6, 2019
Habituation Learning Is a Widely Affected Mechanism in Drosophila Models of Intellectual Disability and Autism Spectrum DisordersMichaela Fenckova, Laura E R Blok, Lenke Asztalos, et al.
Translational Psychiatry|January 8, 2026
Pre-Pulse Inhibition of an escape response in adult fruit fly, Drosophila melanogasterErika Viragh, Lenke Asztalos, Michaela Fenckova, et al.
Plos Genetics|May 12, 2016
BOD1 Is Required for Cognitive Function in Humans and DrosophilaSahar Esmaeeli-Nieh, Michaela Fenckova, Iain M Porter, et al.
Plos One|February 13, 2019
Conserved regulation of neurodevelopmental processes and behavior by FoxP in DrosophilaAnna Castells-Nobau, Ilse Eidhof, Michaela Fenckova, et al.
Plos Genetics|October 26, 2017
Functional convergence of histone methyltransferases EHMT1 and KMT2C involved in intellectual disability and autism spectrum disorderTom S Koemans, Tjitske Kleefstra, Melissa C Chubak, et al.
Human Molecular Genetics|April 12, 2013
CEP89 is required for mitochondrial metabolism and neuronal function in man and flyBregje W M van Bon, Merel A W Oortveld, Leo G Nijtmans, et al.
American Journal of Human Genetics|June 26, 2012
Disruption of an EHMT1-associated chromatin-modification module causes intellectual disabilityTjitske Kleefstra, Jamie M Kramer, Kornelia Neveling, et al.
Journal of Medical Genetics|May 7, 2013
GATAD2B loss-of-function mutations cause a recognisable syndrome with intellectual disability and are associated with learning deficits and synaptic undergrowth in DrosophilaMarjolein H Willemsen, Bonnie Nijhof, Michaela Fenckova, et al.
European Journal of Human Genetics : EJHG|January 14, 2016
De novo loss-of-function mutations in WAC cause a recognizable intellectual disability syndrome and learning deficits in DrosophilaDorien Lugtenberg, Margot R F Reijnders, Michaela Fenckova, et al.
Pageof 3