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Cell Death & Disease|August 14, 2019
Retraction Note: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformationTahereh Javaheri, Zahra Kazemi, Jan Pencik, et al.Molecular Systems Biology|April 24, 2020
STAT3-dependent analysis reveals PDK4 as independent predictor of recurrence in prostate cancerMonika Oberhuber, Matteo Pecoraro, Mate Rusz, et al.Molecular Cancer|November 1, 2024
Cell-autonomous IL6ST activation suppresses prostate cancer development via STAT3/ARF/p53-driven senescence and confers an immune-active tumor microenvironmentChristina Sternberg, Martin Raigel, Tanja Limberger, et al.The Journal of Experimental Medicine|February 24, 2018
ADAM17 is required for EGF-R-induced intestinal tumors via IL-6 trans-signalingStefanie Schmidt, Neele Schumacher, Jeanette Schwarz, et al.Leukemia|August 23, 2018
Dependency on the TYK2/STAT1/MCL1 axis in anaplastic large cell lymphomaNicole Prutsch, Elisabeth Gurnhofer, Tobias Suske, et al.Leukemia|July 16, 2020
Correction: Dependency on the TYK2/STAT1/MCL1 axis in anaplastic large cell lymphomaNicole Prutsch, Elisabeth Gurnhofer, Tobias Suske, et al.Haematologica|April 25, 2020
Whole Exome Sequencing reveals NOTCH1 mutations in anaplastic large cell lymphoma and points to Notch both as a key pathway and a potential therapeutic targetHugo Larose, Nina Prokoph, Jamie D Matthews, et al.Molecular Cancer|May 29, 2024
JUN mediates the senescence associated secretory phenotype and immune cell recruitment to prevent prostate cancer progressionTorben Redmer, Martin Raigel, Christina Sternberg, et al.Haematologica|May 25, 2019
High activation of STAT5A drives peripheral T-cell lymphoma and leukemiaBarbara Maurer, Harini Nivarthi, Bettina Wingelhofer, et al.International Journal of Cancer|October 9, 2020
Thyroid and androgen receptor signaling are antagonized by μ-Crystallin in prostate cancerOsman Aksoy, Jan Pencik, Markus Hartenbach, et al.Pageof 7